Upregulated beta-catenin signaling does not affect survival of pancreatic cancer cells during dual inhibition of GSK3B and HDAC

Nesteruk Kateryna; Smits Ron; Bruno Marco; Peppelenbosch Maikel P.; Fuhler Gwenny M.

首页> 外文期刊>Pancreatology: official journal of the International Association of Pancreatology (IAP) ... [et al.] >Upregulated beta-catenin signaling does not affect survival of pancreatic cancer cells during dual inhibition of GSK3B and HDAC

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Upregulated beta-catenin signaling does not affect survival of pancreatic cancer cells during dual inhibition of GSK3B and HDAC

机译：上调的β-连环蛋白信号在GSK3B和HDAC的双重抑制过程中不会影响胰腺癌细胞的存活

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《Pancreatology: official journal of the International Association of Pancreatology (IAP) ... [et al.]》 |2020年第3期|共4页
作者
Nesteruk Kateryna; Smits Ron; Bruno Marco; Peppelenbosch Maikel P.; Fuhler Gwenny M.;
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作者单位

Erasmus MC Dept Gastroenterol &

Hepatol Rotterdam Netherlands;

Erasmus MC Dept Gastroenterol &

Hepatol Rotterdam Netherlands;

Erasmus MC Dept Gastroenterol &

Hepatol Rotterdam Netherlands;

Erasmus MC Dept Gastroenterol &

Hepatol Rotterdam Netherlands;

Erasmus MC Dept Gastroenterol &

Hepatol Rotterdam Netherlands;

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正文语种 eng
中图分类消化系及腹部疾病;
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1. Upregulated beta-catenin signaling does not affect survival of pancreatic cancer cells during dual inhibition of GSK3B and HDAC [J] . Nesteruk Kateryna, Smits Ron, Bruno Marco, Pancreatology: official journal of the International Association of Pancreatology (IAP) ... [et al.] . 2020,第3期

机译：上调的β-连环蛋白信号在GSK3B和HDAC的双重抑制过程中不会影响胰腺癌细胞的存活
2. Inhibition of EP2/EP4 prostanoid receptor-mediated signaling suppresses IGF-1-induced proliferation of pancreatic cancer BxPC-3 cells via upregulating gamma-glutamyl cyclotransferase expression [J] . Takahashi Tetsuyuki, Ichikawa Hirona, Morimoto Yuuki, Biochemical and Biophysical Research Communications . 2019,第2期

机译：EP2 / EP4前列腺受体介导的信号传导的抑制通过上调γ-谷氨酸循环转移酶表达抑制IGF-1诱导的胰腺癌BXPC-3细胞的增殖
3. Dual inhibition of PI3K/Akt signaling and the DNA damage checkpoint in p53-deficient cells with strong survival signaling: implications for cancer therapy. [J] . Skladanowski A, Bozko P, Sabisz M, Cell cycle . 2007,第18期

机译：p3K / Akt信号转导和p53缺陷细胞DNA损伤检查点的双重抑制，具有强大的存活信号转导：对癌症治疗的意义。
4. Comparison of WI-38 and NIH/3T3 Cells as Standards for the Evaluation of the Inhibition of Pancreatic Cancer Cell Lines by Organotin Polymers Containing Histamine and Phentolamine [C] . Michael R. Roner, Charles E. Carraher, Alisa Moric, American Chemical Society., Division of Polymeric Materials : Science and Engineering., Meeting . 2013

机译：WI-38和NIH / 3T3细胞作为含有组胺和芬兰的有机素聚合物评估胰腺癌细胞系抑制的标准的标准
5. Divergent Regulation and Function of Wnt/beta-Catenin Signaling and TCF Transcription Factors in Pancreatic Cancer [D] . Kershaw, Kathleen. 2018

机译：Wnt /β-Catenin信号传导和TCF转录因子在胰腺癌中的不同调控和功能
6. Pristimerin a quinonemethide triterpenoid induces apoptosis in pancreatic cancer cells through the inhibition of pro-survival Akt/NF-κB/mTOR signaling proteins and anti-apoptotic Bcl-2 [O] . DORRAH DEEB, XIAOHUA GAO, YONG BO LIU, -1

机译：Pristimerin一种醌类三萜类化合物通过抑制促存活的Akt /NF-κB/ mTOR信号蛋白和抗凋亡Bcl-2诱导胰腺癌细胞凋亡。
7. Upregulated β-catenin signaling does not affect survival of pancreatic cancer cells during dual inhibition of GSK3B and HDAC [O] . Kateryna Nesteruk, Ron Smits, Marco Bruno, 2020

机译：上调β-catenin信号传导在GSK3B和HDAC的双重抑制过程中不会影响胰腺癌细胞的存活
8. Inhibition of Histone Deacetylases (HDACs) and mTOR Signaling: Novel Strategies Towards the Treatment of Prostate Cancer. [R] . L. Ellis 2012

机译：抑制组蛋白去乙酰化酶（HDaCs）和mTOR信号：治疗前列腺癌的新策略。

Upregulated beta-catenin signaling does not affect survival of pancreatic cancer cells during dual inhibition of GSK3B and HDAC

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