Regulation of Transforming Growth Factor-#beta# Signaling

Hong-Jian Zhu; Antony W. Burgess

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Regulation of Transforming Growth Factor-#beta# Signaling

机译：转化生长因子-＃beta＃信号传导的调节

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摘要

Members of transforming growth factor #beta# (TGF-#beta#) faily are potent regulators of multiple cellular functions, including cell prolifferatiion, differentiation, migration, organization, and death.

机译：转化生长因子＃beta＃（TGF-＃beta＃）失效的成员是多种细胞功能的有效调节剂，包括细胞增殖，分化，迁移，组织和死亡。

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《Molecular Cell Biology Research Communications》 |2001年第6期|共10页
作者
Hong-Jian Zhu; Antony W. Burgess;
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正文语种 eng
中图分类分子生物学;
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1. Regulation of Transforming Growth Factor-#beta# Signaling [J] . Hong-Jian Zhu, Antony W. Burgess Molecular Cell Biology Research Communications . 2001,第6期

机译：转化生长因子-＃beta＃信号传导的调节
2. Progranulin Promotes Bleomycin-Induced Skin Sclerosis by Enhancing Transforming Growth Factor-beta/Smad3 Signaling through Up-Regulation of Transforming Growth Factor-beta Type I Receptor [J] . Yang Ting, Zhang Xuemei, Chen Aijun, American Journal of Pathology: Official Publication of the American Association of Pathologists . 2019,第8期

机译：通过提高转化生长因子-βα型受体的上调，通过增强转化生长因子-β-β/ smad3信号传导来促进博尔霉素诱导的皮肤硬化。
3. Distinct roles of transforming growth factor-beta signaling and transforming growth factor-beta receptor inhibitor SB431542 in the regulation of p21 expression [J] . Koo Bon-Hun, Kim Yeaji, Cho Yang Je, European Journal of Pharmacology: An International Journal . 2015,第Null期

机译：转化生长因子-β信号转导和转化生长因子-β受体抑制剂SB431542在调节p21表达中的不同作用
4. Extracellular regulation of transforming growth factor β and bone morphogenetic protein signaling in bone [C] . Harikiran Nistala, Sui Lee-Arteaga, Gabriella Siciliano, Conference on Skeletal Biology and Medicine . 2010

机译：骨中生长因子β和骨形态发生蛋白信号传导的细胞外调节
5. Transforming growth factor-beta1 activation and transforming growth factor-beta receptor 2 expression levels in the regulation of the TGF-beta signaling pathway. [D] . Rojas, Andres. 2008

机译：在调节TGF-beta信号通路中转化生长因子-beta1激活和转化生长因子-beta受体2的表达水平。
6. Withdrawal: Cooperative regulation of NOTCH1 protein-phosphatidylinositol 3-kinase (PI3K) signaling by NOD1 NOD2 and TLR2 receptors renders enhanced refractoriness to transforming growth factor-β (TGF-β)- or cytotoxic T-lymphocyte antigen 4 (CTLA-4)-mediated impairment of human dendritic cell maturation. [O] . Devram Sampat Ghorpade, Srini V. Kaveri, Jagadeesh Bayry, 2019

机译：戒断：NOD1NOD2和TLR2受体对NOTCH1蛋白-磷脂酰肌醇3-激酶（PI3K）信号的协同调节可提高对转化生长因子-β（TGF-β）或细胞毒性T淋巴细胞抗原4（CTLA-4）的抵抗力介导的人类树突状细胞成熟受损。
7. Erratum to: Transforming Growth Factor b1/Smad4 Signaling Affects Osteoclast Differentiation via Regulation of miR-155 Expression [O] . 2017

机译：错误至：转化生长因子B1 / Smad4信号传导通过调节MiR-155表达影响骨壳分化
8. Transforming Growth Factor Beta Signaling in Growth of Estrogen- Insensitive Metastatic Bone Lesions. [R] . Nickerson, N. 2012

机译：转化生长因子β信号转导雌激素不敏感转移性骨病变。

Regulation of Transforming Growth Factor-#beta# Signaling

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