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Ultrasound, Clinical, and Electrophysiological Findings in Persistent Carpal Tunnel Syndrome

机译:持久性腕管综合征中的超声波,临床和电生理学发现

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BACKGROUND AND PURPOSE We present the clinical, electrophysiological, and nerve ultrasound findings in cases of persistent carpal tunnel syndrome (PCTS). METHODS Eighteen PCTS patients underwent evaluation with the Boston Carpal Tunnel Syndrome Questionnaire (BCTSQ), electrophysiology, and nerve ultrasound with a mean of 3.5 months (SD +/- 1.4) after open surgery. RESULTS PCTS patients showed a mean symptom severity scale score of 3.1 (SD +/- 1.1) and functional severity scale score of 3.2 (SD +/- 0.9) in BCTSQ. Nerve conduction studies revealed axonal affection of the median nerve in 13/18 patients, ultrasound showed disturbed echogenicity in all patients, a pathological wrist to forearm ratio in 16/18 patients, and cross-sectional area enlargement of the median nerve at the distal wrist crease in 12/18 patients. Ultrasound documented scar tissue formation (in 12/18 patients), incomplete release of retinaculum flexorum (in 4/18 patients), and neuroma of the median nerve (in 2/18 patients) as PCTS cause. CONCLUSION Our data show significant functional disability, axonal nerve damage, and scar tissue formation as common PCTS causes.
机译:背景和目的我们在持续的腕管综合征(PCTS)的情况下介绍临床,电生理学和神经超声检查结果。方法患有18例PCTS患者接受过波士顿腕管综合征问卷调查问卷(BCTSQ),电生理学和神经超声,均为3.5个月(SD +/- 1.4)。结果PCTS患者显示出平均症状严重程度得分为3.1(SD +/- 1.1),并且在BCTSQ中的功能严重比例为3.2(SD +/- 0.9)。神经传导研究揭示了13/18名患者中位神经的轴突情感,超声出现了所有患者的迟交性,一种病理腕部在16/18患者中的前臂比率,以及在远端手腕上的横截面区域扩大了中位神经12/18患者的折痕。超声记录瘢痕组织形成(12/18患者),不完全释放视网膜素柔性(4/18患者),以及中位神经的神经瘤(2/18患者)作为PCTS原因。结论我们的数据显示出显着的功能性残疾,轴突神经损伤和瘢痕组织形成作为普通PCT的原因。

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