机译:通过增强MiR-181D-5P靶向的LRP6和-Catenin介导的OPG来抑制骨髓细胞发生的锶
Jinan Univ Coll Med 2 Shenzhen Peoples Hosp Dept Spine Surg Shenzhen 518000 Peoples R China;
Tianjin Univ Sch Mat Sci &
Engn Tianjin 300072 Peoples R China;
Univ Hong Kong Li Ka Shing Fac Med Dept Orthopaed &
Traumatol Hong Kong Peoples R China;
Univ Hong Kong Li Ka Shing Fac Med Dept Orthopaed &
Traumatol Hong Kong Peoples R China;
Univ Hong Kong Li Ka Shing Fac Med Dept Orthopaed &
Traumatol Hong Kong Peoples R China;
Univ Hong Kong Li Ka Shing Fac Med Dept Orthopaed &
Traumatol Hong Kong Peoples R China;
Univ Hong Kong Li Ka Shing Fac Med Dept Orthopaed &
Traumatol Hong Kong Peoples R China;
Ningxia Med Univ Gen Hosp Dept Traumatol Yinchuan Ningxia Hui Aut Peoples R China;
Univ Hong Kong Li Ka Shing Fac Med Dept Orthopaed &
Traumatol Hong Kong Peoples R China;
Univ Hong Kong Li Ka Shing Fac Med Dept Orthopaed &
Traumatol Hong Kong Peoples R China;
Jinan Univ Coll Med 2 Shenzhen Peoples Hosp Dept Spine Surg Shenzhen 518000 Peoples R China;
Strontium; Osteoclast; MiR-181; Osteoprotegerin; beta-catenin;
机译:通过增强MiR-181D-5P靶向的LRP6和-Catenin介导的OPG来抑制骨髓细胞发生的锶
机译:锶通过增强由miR-181d-5p靶向的LRP6和Catenin介导的OPG来抑制骨核细胞发生
机译:纳米针锶取代的磷灰石涂层通过促进骨质发生和抑制骨质细胞发生来增强骨质疏松骨整合
机译:OPGL是破骨细胞生成和骨骼的关键调节剂形态发生
机译:用CNT-STM尖端在HOPG中的镧锶锰氧化物和蜂窝晶格中进行扫描隧道显微镜。
机译:锶通过增强miR-181d-5p靶向的LRP6和β-catenin介导的OPG抑制破骨细胞生成
机译:骨酸溶胶发生抑制因子(OCIF)和骨盆素(OPG)的特性:OPG / OCIF在体外抑制骨细胞发生的机制