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机译:饱和脂肪酸通过Toll样受体4 / NF-Kappab信号传导激活微胶质细胞。
Shandong Provincial Key Laboratory of Mental Disorders Department of Histology and Embryology Shandong University School of Medicine 44# Wenhua Xi Road Jinan Shandong 250012 People's Repubic of China;
Department of Anatomy Yong Loo Lin School of Medicine National University of Singapore 4 Medical Drive MD10 Singapore 117597 Singapore;
Shandong Provincial Key Laboratory of Mental Disorders Department of Histology and Embryology Shandong University School of Medicine 44# Wenhua Xi Road Jinan Shandong 250012 People's Repubic of China;
Department of Anatomy Yong Loo Lin School of Medicine National University of Singapore 4 Medical Drive MD10 Singapore 117597 Singapore;
Shandong Provincial Key Laboratory of Mental Disorders Department of Histology and Embryology Shandong University School of Medicine 44# Wenhua Xi Road Jinan Shandong 250012 People's Repubic of China;
Department of Anatomy Yong Loo Lin School of Medicine National University of Singapore 4 Medical Drive MD10 Singapore 117597 Singapore;
Shandong Provincial Key Laboratory of Mental Disorders Department of Histology and Embryology Shandong University School of Medicine 44# Wenhua Xi Road Jinan Shandong 250012 People's Repubic of China;
Microglia; SFA; NF-κB; Toll-like receptor 4;
机译:饱和脂肪酸通过Toll样受体4 / NF-Kappab信号传导激活微胶质细胞。
机译:饱和脂肪酸激活Toll样受体2/4和二十二碳六烯酸抑制的机制
机译:激活转录因子3构成了一个负反馈机制,该机制减弱了肥胖脂肪组织中饱和脂肪酸/ toll样受体4的信号传导和巨噬细胞的激活。
机译:线粒体的遗传障碍!和过氧化物血型脂肪酸氧化和过氧化物体增殖活化受体
机译:肥胖引起的炎症和胰岛素抵抗的发展:脂肪组织,脂肪酸和Toll样受体的作用。
机译:饱和脂肪酸激活Toll样受体2/4和二十二碳六烯酸抑制的机制
机译:饱和脂肪酸激活,但多不饱和脂肪酸抑制与Toll样受体6或1二聚化的Toll样受体2
机译:定义膳食脂肪酸在乳腺癌中的分子作用:过氧化物酶体增殖物激活的受体γ的选择性调节。附录;最终的评论。 2006年9月30日至2008年4月8日