机译:CLC5通过促进刺激自噬降低多发性骨髓瘤细胞对硼替佐米的敏感性
Tianjin Med Univ Dept Hematopathol Gen Hosp 154 Anshan Rd Tianjin 300052 Peoples R China;
Shijiazhuang Pingan Hosp Dept Hematol Shijiazhuang Hebei Peoples R China;
Shijiazhuang Pingan Hosp Dept Hematol Shijiazhuang Hebei Peoples R China;
Shijiazhuang Pingan Hosp Dept Hematol Shijiazhuang Hebei Peoples R China;
Tianjin Med Univ Dept Hematopathol Gen Hosp 154 Anshan Rd Tianjin 300052 Peoples R China;
Tianjin Med Univ Dept Hematopathol Gen Hosp 154 Anshan Rd Tianjin 300052 Peoples R China;
Bortezomib (BZ); Multiple myeloma; Chemoresistance; Autophagy; ClC5; AKT-mTOR pathway;
机译:CLC5通过促进刺激自噬降低多发性骨髓瘤细胞对硼替佐米的敏感性
机译:BRD4抑制剂NITROXOLINE通过促进线粒体途径介导的细胞凋亡,增强了多发性骨髓瘤细胞对硼替佐米的敏感性和体内的敏感性
机译:线粒体调节降低多发性骨髓瘤细胞对硼替佐米的耐药性
机译:Bortezomib抗性多发性骨髓瘤的初步大规模定量蛋白质组学分析
机译:Ixazomib的第1/2期作为替代品髓鞘或胭脂瘤患者的替代品的替代物,最近对含有Bortezomib或Carfilzomib的最后组合方案复发或难治
机译:MiR145-3P通过靶向HDAC4促进自噬并增强多发性骨髓瘤中的硼脲敏感性
机译:BRD4抑制剂NITROXOLINE通过促进线粒体途径介导的细胞凋亡,增强了多发性骨髓瘤细胞对硼替佐米的敏感性和体内的敏感性