机译:亚磺烷恢复乙酰基组蛋白H3与Bcl-2启动子结合,并防止乙醇暴露的神经顶部细胞和小鼠胚胎中的细胞凋亡
Department of Pharmacology and Toxicology University of Louisville Health Sciences Center;
Department of Pharmacology and Toxicology University of Louisville Health Sciences Center;
Department of Pharmacology and Toxicology University of Louisville Health Sciences Center;
University of Louisville Alcohol Research Center;
Pediatric Research Institute Department of Pediatrics University of Louisville;
Ben May Department for Cancer Research University of Chicago;
Department of Pharmacology and Toxicology University of Louisville Health Sciences Center;
Sulforaphane; Ethanol; Apoptosis; Histone deacetylase; Bcl-2;
机译:亚磺烷恢复乙酰基组蛋白H3与Bcl-2启动子结合,并防止乙醇暴露的神经顶部细胞和小鼠胚胎中的细胞凋亡
机译:通过在表现蜗牛的表达1通过恢复上皮 - 间充质转换来恢复上皮 - 间充质转换来保护亚氟烃在神经嵴细胞中防止乙醇诱导的细胞凋亡
机译:乙醇暴露的小鼠神经Mouse细胞中铁介导的自由基损伤
机译:使用卷积神经网络自动预测早期小鼠和人类胚胎的细胞阶段
机译:发育中的小鼠胚胎中神经c细胞的诱导和模式:GCNF和HHAT的作用
机译:萝卜硫素可恢复乙酰组蛋白H3与Bcl-2启动子的结合并防止乙醇暴露的神经cells细胞和小鼠胚胎发生凋亡
机译:亚氟烃通过在编码凋亡蛋白抑制剂的基因的启动子下递减乙醇诱导的高甲基化,防止乙醇诱导的人神经嵴细胞凋亡