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机译:AMPK编排了一种精心级联保护组织免受纤维化和老化
Northwest Univ Xian Key Lab Resource Biol &
Biotechnol Western China Minist Educ Fac Life Sci;
Fourth Mil Med Univ Dept Biomed Engn 169 Changle West Rd Xian 710032 Shaanxi Peoples R China;
Fourth Mil Med Univ Dept Biomed Engn 169 Changle West Rd Xian 710032 Shaanxi Peoples R China;
Fourth Mil Med Univ Xijing Hosp Dept Cardiovasc Surg 127 Changle West Rd Xian 710032 Shaanxi;
Fourth Mil Med Univ Tangdu Hosp Dept Thorac Surg 1 Xinsi Rd Xian 710038 Shaanxi Peoples R;
Fourth Mil Med Univ Xijing Hosp Dept Geriatr 127 Changle West Rd Xian 710032 Shaanxi Peoples;
Nanjing Univ Med Sch Affiliated Drum Tower Hosp Dept Thorac &
Cardiovasc Surg 321 Zhongshan Rd;
Northwest Univ Xian Key Lab Resource Biol &
Biotechnol Western China Minist Educ Fac Life Sci;
AMP-activated protein kinase; Fibrosis; Fibrogenesis; Aging; Inflammation; Excessive extracellular matrix;
机译:AMPK编排了一种精心级联保护组织免受纤维化和老化
机译:通过抑制携带人组织激肽释放酶1的老年转基因大鼠的Rho激酶/ LIM激酶/ cofilin途径,减少体质纤维化以保护勃起功能
机译:132减少体纤维化以通过抑制患者携带人体组织Kallikrein的老年转基因大鼠中的Smad和非Smad途径来保护勃起功能
机译:AMPK信号级联参与辣椒素诱导的HT-29结肠癌细胞凋亡。
机译:AMPK功能以非自主方式调节组织和生物衰老
机译:通过抑制携带人组织激肽释放酶1的衰老转基因大鼠的Rho激酶/ LIM激酶/ cofilin途径减少体质纤维化以保护勃起功能
机译:巨噬细胞和纤维化:单核吞噬细胞的驻留和浸润如何协调组织损伤和修复的所有阶段