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机译:PGAM5-CYPD途径涉及溴杉裂性诱导的RIP3 / MLKL依赖性腐败性腐败症状细胞
Army Med Univ Multidisciplinary Ctr Pituitary Adenomas Chongqin Dept Neurosurg Xinqiao Hosp;
Xi An Jiao Tong Univ Dept Lab Med Xian Cent Hosp 161 Xi Wu Rd Xian 710003 Shaanxi Peoples R;
Army Med Univ Multidisciplinary Ctr Pituitary Adenomas Chongqin Dept Neurosurg Xinqiao Hosp;
Army Med Univ Multidisciplinary Ctr Pituitary Adenomas Chongqin Dept Neurosurg Xinqiao Hosp;
Army Med Univ Multidisciplinary Ctr Pituitary Adenomas Chongqin Dept Neurosurg Xinqiao Hosp;
Army Med Univ Multidisciplinary Ctr Pituitary Adenomas Chongqin Dept Neurosurg Xinqiao Hosp;
Army Med Univ Multidisciplinary Ctr Pituitary Adenomas Chongqin Dept Neurosurg Xinqiao Hosp;
Prolactinoma; Necroptosis; Bromocriptine; MMQ cells;
机译:PGAM5-CYPD途径涉及溴杉裂性诱导的RIP3 / MLKL依赖性腐败性腐败症状细胞
机译:通过靶向RIP1 / RIP3 / PGAM5途径,Bufalin在乳腺癌细胞中参与RIP1依赖性和ROS依赖性编程的粪便
机译:大黄素通过TNF-α/ RIP1 / RIP3途径诱导胶质瘤细胞系U251中的粪便
机译:灌注细胞培养揭示帕拉卡碱或自分泌信号通路,参与脂肪衍生的干细胞分化成脂肪细胞
机译:通过氧化应激和病毒感染引起的细胞死亡途径裸鼠抑菌介导机制
机译:RIP3的上调3通过激活RIP3 / MLK信号通路和抗病症的诱导来减轻前列腺癌进展
机译:各种触发因子与缺陷症中的rip1 / RIP3信号通路之间的连接