机译:T细胞触发阈值由单个T细胞受体簇中抗原的数量调节
Howard Hughes Medical Institute, Department of Chemistry, ,Biophysics Graduate Group, University of California, Berkeley, CA 94720;
Howard Hughes Medical Institute, Department of Chemistry, ,Physical Biosciences and Materials Sciences Divisions, Lawrence Berkeley National Laboratory, Berkeley, CA 94720;
Howard Hughes Medical Institute, Department of Chemistry, ,Physical Biosciences and Materials Sciences Divisions, Lawrence Berkeley National Laboratory, Berkeley, CA 94720;
Program in Molecular Pathogenesis, Skirball Institute of Biomolecular Medicine and Department of Pathology, New York University School of Medicine, New York, NY 10016;
Howard Hughes Medical Institute, Department of Chemistry, ,Biophysics Graduate Group, University of California, Berkeley, CA 94720,Physical Biosciences and Materials Sciences Divisions, Lawrence Berkeley National Laboratory, Berkeley, CA 94720;
cell biophysics; cell patterning; immune synapse;
机译:T细胞抗原受体触发和脂质筏:一个时空尺度问题–关于脂质筏参与T细胞活化的话题
机译:细胞死亡的易感性是显性表型:触发活化驱动的T细胞死亡,独立于T细胞抗原受体复合物。
机译:尽管超过了胸腺选择阈值,但用于肿瘤/自身抗原的增强亲和力的鼠T细胞受体在基因治疗中仍是安全的。
机译:磷脂酶Cgammal耦合到B-和T细胞抗原受体的机制
机译:在内质网中保留T细胞抗原受体α链变体的结构和功能决定因素。
机译:T细胞触发阈值由单个T细胞受体簇中抗原的数量调节
机译:T细胞触发阈值由单个T细胞受体簇中抗原的数量调节。