机译:肺癌进展过程中对p53恢复的阶段特异性敏感性
Koch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnTufts University, and Harvard Medical School, 77 Avenue Louis Pasteur, Boston, Massachusetts 02115, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
rnKoch Institute for Integrative Cancer Research, Departmentof Biology, and Howard Hughes Medical Institute, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, Massachusetts 02139, USA;
机译:P53R2,p53诱导型核糖核苷酸还原酶基因与非小细胞肺癌的肿瘤进展有关。
机译:野生型p53基因转染p53无效的人非小细胞肺癌H358细胞后,诱导衰老样表型和紫杉醇敏感性丧失。
机译:在进展中检测组织或液体活检组织中TP53突变可确定ALK +肺癌患者生存率较低
机译:从审查的异质性临床成像对肺癌和无癌进展的深度多任务预测
机译:使用重组突变体/野生型p53杂合性人类乳腺上皮细胞培养模型确定p53突变在人类乳腺癌进展中的作用。
机译:肺癌进展期间的p53恢复阶段特异性灵敏度
机译:在肺癌进展期间对p53恢复的阶段特异性敏感性