机译:通过破坏激活剂-介体相互作用抑制真菌多药耐药性
Massachusetts Gen Hosp, Ctr Canc, Charlestown, MA 02129 USA|Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dept Biol Chem & Mol Pharmacol, Boston, MA 02115 USA;
Univ Lausanne Hosp, Inst Microbiol, CH-1011 Lausanne, Switzerland|Univ Hosp Ctr, CH-1011 Lausanne, Switzerland;
Univ Cattolica Sacro Cuore, Inst Microbiol, I-00168 Rome, Italy;
Univ Cattolica Sacro Cuore, Inst Publ Hlth, I-00168 Rome, Italy;
Massachusetts Gen Hosp, Ctr Canc, Charlestown, MA 02129 USA|Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA;
Massachusetts Gen Hosp, Dept Mol Biol, Boston, MA 02114 USA|Harvard Univ, Sch Med, Dept Genet, Boston, MA 02115 USA;
Univ Sofia, Fac Chem & Pharm, Sofia 1164, Bulgaria;
Univ Lausanne Hosp, Inst Microbiol, CH-1011 Lausanne, Switzerland|Univ Hosp Ctr, CH-1011 Lausanne, Switzerland;
Univ Cattolica Sacro Cuore, Inst Microbiol, I-00168 Rome, Italy;
Massachusetts Gen Hosp, Dept Mol Biol, Boston, MA 02114 USA|Massachusetts Gen Hosp, Dept Pathol, Boston, MA 02114 USA|Harvard Univ, Sch Med, Boston, MA 02114 USA;
Indian Inst Technol Delhi, Dept Chem, New Delhi 110016, India|Indian Inst Technol Delhi, Supercomp Facil Bioinformat & Computat Biol, New Delhi 110016, India;
Indian Inst Technol Delhi, Dept Chem, New Delhi 110016, India|Indian Inst Technol Delhi, Supercomp Facil Bioinformat & Computat Biol, New Delhi 110016, India|Indian Inst Technol Delhi, Kusuma Sch Biol Sci, New Delhi 110016, India;
Harvard Univ, Sch Med, Dept Biol Chem & Mol Pharmacol, Boston, MA 02115 USA|Dana Farber Canc Inst, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dept Biol Chem & Mol Pharmacol, Boston, MA 02115 USA|Dana Farber Canc Inst, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dept Biol Chem & Mol Pharmacol, Boston, MA 02115 USA;
Massachusetts Gen Hosp, Ctr Canc, Charlestown, MA 02129 USA|Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dept Biol Chem & Mol Pharmacol, Boston, MA 02115 USA;
机译:PDE5抑制剂西地那非和伐地那非可通过抑制多药耐药蛋白7(ATP结合盒式C10)转运蛋白的外排功能逆转多药耐药性
机译:PDE5抑制剂sildenafil和vardenafil可通过抑制多重耐药蛋白7(ATP结合盒式C10)转运蛋白的外排功能逆转多重耐药
机译:细胞周期蛋白依赖性激酶抑制剂AT-7519,flavopiridol和SNS-032与ABCB1,ABCG2和ABCC1转运蛋白的相互作用及其在体外克服多药耐药性的潜力
机译:真菌相互作用的MALDI成像:一种促进从海洋细菌中脱离真菌抑制剂的工具
机译:金纳米晶体治疗:耐多药病原体的治疗和破坏蛋白质/蛋白质相互作用。
机译:通过破坏激活剂-介体相互作用来抑制真菌的多药耐药性
机译:通过破坏激活剂-介体相互作用抑制真菌多药耐药性
机译:发现有效抑制剂对乳腺癌多药耐药性的影响