机译:铁氧化还原化学促进α-突触核蛋白的反平行低聚
Department of Chemistry, Virginia Commonwealth University, Richmond, Virginia 23284, United States;
Department of Chemistry, Virginia Commonwealth University, Richmond, Virginia 23284, United States;
Department of Chemistry, Virginia Commonwealth University, Richmond, Virginia 23284, United States;
Department of Chemistry, Virginia Commonwealth University, Richmond, Virginia 23284, United States;
Department of Chemistry, Virginia Commonwealth University, Richmond, Virginia 23284, United States;
Department of Chemistry, Virginia Commonwealth University, Richmond, Virginia 23284, United States;
Department of Chemistry, Virginia Commonwealth University, Richmond, Virginia 23284, United States;
机译:氧化铜纳米粒子促进α-突触核蛋白寡聚化和神经毒性作为帕金森病的模型
机译:多不饱和脂肪酸衍生的脂质过氧化产物对α-突触核蛋白的修饰可促进毒性低聚:其与帕金森氏病的相关性
机译:通过衍生自多不饱和脂肪酸的脂质过氧化产物的α-突触核蛋白的修饰促进了毒性寡聚:其与帕金森病的相关性
机译:含铁矿物和EDTA存在下的Pu Redox化学
机译:研究与环境有关的氧化铁和硫化物的表面氧化还原化学。
机译:α-突触核蛋白低聚增加其促进NMDA受体内化的作用
机译:低摩尔过量的4-氧代-2-壬酯和4-羟基-2-诺纳通过不同的途径促进α-突触核蛋白的低聚