机译:胰腺癌的器官型培养模型表明基质细胞调节肿瘤细胞中的E-钙黏着蛋白,β-连环蛋白和Ezrin表达。
From the Centre for Tumour Biology,Institute of Cancer, Barts and the London School of Medicine and Dentistry, London;
From the Centre for Tumour Biology,Institute of Cancer, Barts and the London School of Medicine and Dentistry, London;
the Departments of Pathology,Barts and the London Hepato-Pancreatico-Biliary Centre, Royal London Hospital, London;
and the Histopathology Unit,Cancer Research UK, London, United Kingdom;
and Surgery,Barts and the London Hepato-Pancreatico-Biliary Centre, Royal London Hospital, London;
From the Centre for Tumour Biology,Institute of Cancer, Barts and the London School of Medicine and Dentistry, London;
From the Centre for Tumour Biology,Institute of Cancer, Barts and the London School of Medicine and Dentistry, London|and Surgery,Barts and the London Hepato-Pancreatico-Biliary Centre, Royal London Hospital, London;
机译:胰腺癌的器官型培养模型表明,基质细胞调节肿瘤细胞中的E-cadherin,β-catenin和Ezrin表达。
机译:E-钙黏着蛋白/β-连环蛋白在胰腺癌细胞中的表达恢复通过诱导凋亡来抑制生长。
机译:颜色编码的腔内成像显示转化生长因子-β(TGF-β)拮抗剂在人胰腺癌原子小鼠模型中选择性地靶向基质细胞
机译:Wnt /β-catenin途径在单层培养和3D球体中脂肪衍生干细胞成骨分化过程中的激活
机译:Ptf1a-Cre胰腺上皮内瘤变细胞中尿液,粪便和血清样品和胰腺肿瘤的代谢谱分析和HMGA1表达水平的评估; LSL-KrasG12D胰腺癌转基因小鼠模型
机译:胰腺癌的器官型培养模型表明基质细胞调节肿瘤细胞中的E-钙黏着蛋白β-连环蛋白和Ezrin表达。
机译:胰腺癌的器官型培养模型表明基质细胞调节肿瘤细胞中的E-钙粘蛋白,β-连环蛋白和Ezrin表达。