A method for solubilization of hexamethylene tetramine silver salicylate in water

• 1. 从己二腈生产六亚甲基二胺和氨基己腈的方法,其中六亚甲基二胺包含少于200PPM的四氢氮杂 [P] . 中国专利： CN100361961C . 2008.01.16
• 2. 从己二腈生产六亚甲基二胺和氨基己腈的方法,其中六亚甲基二胺包含少于200PPM的四氢氮杂 [P] . 中国专利： CN1852889A . 2006-10-25
• 3. METHOD OF HEXAMETHYLENE-TETRAMINE BIOLOGICAL REMOVAL FROM WASTE WATERS [P] . CS9000872A2 . 1991-12-17

  机译： 去除废水中六亚甲基四丁烯的方法

• 4. METHOD OF HEXAMETHYLENE-TETRAMINE BIOLOGICAL REMOVAL FROM WASTE WATERS [P] . CS87290A2 . 1991-12-17

  机译： 去除废水中六亚甲基四丁烯的方法

• 5. Process for the preparation of a suitable aqueous to injections of hexamethylene tetramine - silver nitrate solution [P] . 德国专利： DE458837C . 1928-04-19

  机译： 制备适合注射六亚甲基四胺-硝酸银溶液的水溶液的方法

• 1. Antimicrobial Effect of Silver Ionised Water Prepared with the Sintering Coating Method—Effect on Acid Production in Plaque [J] . Shiho Morishita, Shigeru Watanabe, Kei Miyazawa, 健康（英文） . 2021,第003期
• 2. Qualitative and Comparative Study of Different Methods of Interpolation for the Mapping of Groundwater Salinity: Case Study of Thermal Waters Used for Irrigation in Northeastern Algeria [J] . Touhami Feraga, Séverin Pistre 地球科学和环境保护期刊（英文） . 2021,第007期
• 3. Improved Methods for the Extraction and Purification of Corynetoxins for Preparative or Analytical Purposes using Water Solubilizing Additives [C] . N. Anderton, Y. Cao, S.M. Colegate, International Symposium on Poisonous Plants . 2004
• 4. Advanced icp-ms methods for examining the stability of silver nanoparticles in natural waters [D] . Gately, Thomas Joseph. 2013
• 5. New method for sequestration of silver nanoparticles in aqueous media: in route toward municipal wastewater [O] . Marie-Laine Roy, Christian Gagnon, Jonathan Gagnon 2016
• 6. Fluorescent X-ray analysis of trace amount of chloride ion in river and rain waters etc. by silver bromide coprecipitation method. [O] . Futoshi SUGIMOTO, Hiroki IWAYA, Yoshimichi MAEDA, 1990
• 7. Methods for Spawning, Culturing and Conducting Toxicity-Tests with Early Life Stages of Four Atherinid Fishes: The Inland Silverside, 'Menidia beryllina', Atlantic Silverside, 'M. menidia', Tidewater Silverside, 'M. peninsulae' and California Grunion, 'Leuresthes tenuis' [R] . Middaugh, D. P. , Hemmer, M. J. , Goodman, L. K. 1987

• 1. A New Three-dimensional Network Constructed by Heptamolybdate, Sodium Ions and Hexamethylene Tetramine Cations via Hydrogen Bonds [J] . 杨文斌 ,卢灿忠 ,庄鸿辉 . 结构化学 . 2002,第002期
• 2. Waterborne Polyurethane-Polyacrylic Ester Hybrid Dispersion Modified by Hexamethylene Diisocyanate Trimer [J] . . 华东理工大学学报（自然科学版） . 2011,第004期
• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. 六亚甲基四胺胺锑(Ⅲ)、铋(Ⅲ)固体配合物的合成与表征 [C] . . 第四届全国配位化学会议 . 2001
• 5. 金属--六亚甲基四胺配位网络衍生的碳/金属碲化物复合材料：制备、界面设计及储钠应用 [A] . 孙典鼎 . 2020