Increase of the water resistance of chrome-tanned or chromium combination-tanned leathers

• 1. 一种利用鞣革铬泥制备碱式硫酸铬鞣液的方法 [P] . 中国专利： CN102965455B . 2014.05.07
• 2. 一种利用鞣革铬泥制备碱式硫酸铬鞣液的方法 [P] . 中国专利： CN102965455A . 2013-03-13
• 3. process for increasing the water resistance of chromium or kombininert chrome - tanned leather [P] . AT201224B . 1958-12-10

  机译： 铬或康比宁铬的耐水性的改进方法-鞣制皮革

• 4. ECO-FRIENDLY LEATHER PACKING CAPABLE OF INCREASING OIL RESISTANCE, CHEMICAL RESISTANCE, ELASTICITY AND WATER RESISTANCE, ETC, AND A ECO-TYPE LEATHER PACKING MANUFACTURED BY THE SAME [P] . 韩国专利： KR20120124183A . 2012-11-13

  机译： 能够提高耐油性，耐化学性，弹性和耐水性，ETC的生态友好型皮革包装，以及由同一制造商生产的生态型皮革包装

• 5. process for obtaining protein and chromium salt by chemical treatment of chrome-tanned leather tailings by countercurrent reactions [P] . BRPI1100438A2 . 2013-04-30

  机译： 逆流化学处理鞣制皮革尾矿获得蛋白质和铬盐的方法

• 1. Increased resistance of drought by Trichoderma harzianum fungal treatment correlates with increased secondary metabolites and proline content [J] . Mona S. Alwhibi, Abeer Hashem12, Elsayed Fathi Abd_Allah, 农业科学学报：英文版 . 2017,第008期
• 2. Removal of Chromium Ions from a Real Wastewater of Leather Industry Using Electrocoagulation and Reverse Osmosis Processes [C] . Rasha H. Salman, Hassanain A. Hassan, Khalid M. Abed, International Conference on Materials Engineering and Science . 2020
• 3. Does the Disinfection of Public Water Supplies Increase Antibiotic Resistance Levels? [D] . ?O'Leary, Emma Jancia 2020
• 4. Increased insulin resistance is associated with increased urinary excretion of chromium in non-diabetic normotensive Saudi adults [O] . Suhad M. Bahijri, Eman M. Alissa 2011
• 5. Chromium (III) Removal from Synthetic Wastewater Using Biochar Produced from Vegetable Tanned Leather Shaving Dust [O] . Tasrina R. Choudhury, U. H. Bodrun Naher, Sarifa Akter, 2020

• 1. The influence of old leather tannery district on chromium contamination of soils,water and plants [J] . Agnieszka Wolińska ,Zofia Stepniewska ,Rafal Wlosek . 自然科学期刊（英文） . 2013,第2期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. Eco-friendly Leather： Chromium Reduction in the Tanning Cycle [J] . Elena Salernitano1 ,Alessandra Strafella1 ,Mercedes Roig2 . 环境科学与工程：A . 2017,第008期
• 4. Spectrophotometric Determination of Water-Soluble Hexavalent Chromium and Determination of Total Hexavalent Chromium Content of Portland Cement in the Presence of Iron (III) and Titanium (IV) Using Derivative Ratio Spectrophotometry [J] . K. A. Idriss ,H. Sedaira ,S. Dardeery . 美国分析化学（英文） . 2013,第11期
• 5. 轻革少铬鞣制工艺研究-植-铬-铝-醛结合鞣 [C] . 程凤侠 ,杨宗邃 ,汪建根 . 第四届亚洲国际皮革科学技术会议 . 1998
• 6. 乙烯基类单体改性水解铬鞣革屑制备蛋白复鞣填充剂的研究 [A] . 陈柱平 . 2011