DEVICE FOR DETERMINING ONE-DIMENSIONAL K-TH ORDER INITIAL MOMENTS OF RANDOM SIGNAL

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• 3. DEVICE FOR DETERMINING SINGLE-DIMENSIONAL INITIAL K-TH ORDER OF RANDOM SIGNAL [P] . SU734721A1 . 1980-05-15

  机译： 确定随机信号的第K维阶次的装置

• 4. ADAPTIVE DEVICE FOR DETERMINING ONE-DIMENSIONAL INITIAL M-TH ORDER MOMENTS OF RANDOM PROCESS [P] . SU684555A1 . 1979-09-05

  机译： 确定一维初始M阶随机过程矩的自适应装置

• 5. DEVICE FOR DETERMINING ONE-DIMENSIONAL INITIAL MTH ORDER MOMENTS OF RANDOM PROCESS [P] . SU590755A1 . 1978-01-30

  机译： 确定一维初始Mth阶随机过程的装置

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• 1. Randomized study comparing incidence of radial artery occlusion post-percutaneous coronary intervention between two conventional compression devices using a novel airinflation technique [J] . Victor Voon ,Muhammad Ayyaz Ul Haq ,Ciara Cahill . 世界心脏病学杂志：英文版（电子版） . 2017,第11期
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• 3. Skewness Based Elman Recurrent Neural Network Model for Classification of Cavitation Signals from Pressure Drop Devices of Prototype Fast Breeder Reactor [J] . Ramadevi Rathinasabapathy ,Sheela Rani Balasubramaniam ,Prakash Vasudevan . 通讯和计算机 . 2011,第007期
• 4. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
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