Press to Talk (PTT) and Press Again to Listen (PATL) Radio/ECM Control

• 1. 通话控制装置、PTT终端及PTT通话系统 [P] . 中国专利： CN202602899U . 2012.12.12
• 2. 一种MCPTT架构下控制信令传输方法以及相关设备 [P] . 中国专利： CN106068625A . 2016-11-02
• 3. Press to Talk (PTT) and Press Again to Listen (PATL) Radio / ECM Control [P] . 英国专利： GB2458143A . 2009-09-09

  机译： 按下通话（PTT），然后再次按下以收听（PATL）收音机/ ECM控制

• 4. Press to talk (ptt) and press again to listen (patl) switch [P] . 英国专利： GB0804161D0 . 2008-04-16

  机译： 按通话（ptt），然后再按一次以收听（patl）开关

• 5. Radio system with a press to talk (PTT) switch forming a part of a switch unit which is supported by the radio [P] . NZ519440A . 2004-03-26

  机译： 具有即按即说（PTT）开关的无线电系统，该系统构成了受无线电支持的开关单元的一部分

• 1. Differential Hydrocarbon Enrichment and its Main Controlling Factors in Depressions of the Bohai Bay Basin Differential Hydrocarbon Enrichment and its Main Controlling Factors in Depressions of the Bohai Bay Basin [J] . JIANG Youlu, LIU Hua, SONG Guoqi, 地质学报（英文版） . 2017,第005期
• 2. Talk Radio Listening, Opinion Expression and Political Discussion in a Democratizing Society [J] . Francis L. F. Lee Asian Journal of Communication . 2007,第1期
• 3. What to listen for in the consultation. Breast cancer patients' own focus on talking about acceptance-based psychological coping predicts decreased psychological distress and depression [J] . JensenC.G., ElsassP., NeustrupL., Patient education and counseling . 2014,第2期
• 4. Talk radio as the soundtrack of our lives: Participatory HIV/AIDS communication, public self-expression and Positive Talk [J] . Mariekie Burger SAHARA-J . 2015,第1期
• 5. Interference suppression in communication systems with controlled crosstalk [C] . Gao, W., Cho, . 1999
• 6. Implementation of a rate control algorithm for compressed sensed videos on a software defined radio. [D] . Prasanna, Arvind. 2011
• 7. CCN4/WISP1 controls cutaneous wound healing by modulating proliferation migration and ECM expression in dermal fibroblasts via α5β1 and TNFα [O] . Mitsuaki Ono, Asuka Masaki, Azusa Maeda, -1
• 8. Listening while talking: The retention of prose under articulatory suppression in relation to simultaneous interpreting [O] . 2006

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Changes of ECM and CAM gene expression profile in the cirrhotic liver after HCV infection: Analysis by cDNA expression array [J] . XinXu ,Yi-MingLi ,HongJi . 世界胃肠病学杂志：英文版 . 2005,第14期
• 3. HMM-Based Photo-Realistic Talking Face Synthesis Using Facial Expression Parameter Mapping with Deep Neural Networks [J] . Kazuki Sato ,Takashi Nose ,Akinori Ito . 电脑和通信（英文） . 2017,第10期
• 4. Concentric Annular Clearance-Controllable Pressure Characteristics Research of a High Pressure Pneumatic Pressure Assembly [J] . WANG Xuanyin ,XU Zhipeng . 中国机械工程学报 . 2011,第001期
• 5. 全台IP数字通话架构下交互演播室通话系统设计 [C] . Cheng Chen ,程晨 ,Li Ningbin . 2017年中国电影电视技术学会城市电视台技术分会科技创新优秀论文评审会 . -1
• 6. 顺应理论和合作原则视角下的无线电陆空通话英语研究 [A] . 林巧彬 . 2013