机译:人类肿瘤基因组学和斑马鱼建模确定SPRED1丢失是粘膜黑色素瘤的驱动因素
Harvard Univ, Harvard Stem Cell Inst, Cambridge, MA 02138 USA;
Harvard Univ, Boston, MA 02115 USA;
Fox Chase Canc Ctr, 7701 Burholme Ave, Philadelphia, PA 19111 USA;
Univ Calif San Francisco, Dept Dermatol, San Francisco, CA 94143 USA;
Boston Childrens Hosp, Div Hematol Oncol, Boston, MA 02115 USA;
Univ Calif San Francisco, Helen Diller Family Comprehens Canc Ctr, San Francisco, CA 94158 USA;
Boston Childrens Hosp, Stem Cell Program, Boston, MA 02115 USA;
Stanford Univ, Dept Med, Med Ctr, Stanford, CA 94305 USA;
Dana Farber Canc Inst, Boston, MA 02115 USA;
Univ Calif San Francisco, Dept Orofacial Sci, San Francisco, CA 94143 USA;
Boston Childrens Hosp, Howard Hughes Med Inst, Boston, MA 02115 USA;
Brigham & Womens Hosp, Dept Pathol, 75 Francis St, Boston, MA 02115 USA;
Univ Calif San Francisco, Dept Pathol, San Francisco, CA 94143 USA;
机译:Spred1是粘膜黑素瘤的肿瘤抑制剂
机译:协同靶向抑制MEK和双重PI3K / mTOR降低了犬黑色素瘤的生存能力并抑制了肿瘤的生长,从而突出了其作为人类粘膜黑色素瘤的临床前模型的效用
机译:海龟纤维乳头状瘤肿瘤与人类癌症共享基因组驱动因素和治疗脆弱性
机译:转移结直肠癌(MCRC)的循环肿瘤细胞(CTC)的单细胞基因组分析识别肿瘤异质性和稀有躯体驾驶员改变
机译:模拟人类肿瘤的进展-识别参与上皮癌变的细胞外基质相互作用网络。
机译:人类肿瘤基因组学和斑马鱼建模确定SPRED1丢失是粘膜黑色素瘤的驱动因素
机译:人和斑马鱼肿瘤中拷贝数变化的比较肿瘤基因组学分析可发现癌症驱动程序