机译:与TET2缺乏症相关的克隆性造血作用加速小鼠的动脉粥样硬化发展
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Yale Univ, Sch Med, Yale Cardiovasc Res Ctr, Vasc Biol & Therapeut Program, 333 Cedar St, New Haven, CT 06511 USA|Yale Univ, Sch Med, Dept Med, 333 Cedar St, New Haven, CT 06511 USA|Yale Univ, Sch Med, Dept Pharmacol, 333 Cedar St, New Haven, CT 06511 USA;
Yale Univ, Sch Med, Yale Cardiovasc Res Ctr, Vasc Biol & Therapeut Program, 333 Cedar St, New Haven, CT 06511 USA|Yale Univ, Sch Med, Dept Med, 333 Cedar St, New Haven, CT 06511 USA|Yale Univ, Sch Med, Dept Pharmacol, 333 Cedar St, New Haven, CT 06511 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Ctr Nacl Invest Cardiovasc Carlos III CNIC, Madrid, Spain|CIBER Enfermedades Cardiovasc, Madrid, Spain;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
Univ Queensland, Inst Mol Biosci, Brisbane, Qld, Australia;
Univ Queensland, Inst Mol Biosci, Brisbane, Qld, Australia;
Ctr Nacl Invest Cardiovasc Carlos III CNIC, Madrid, Spain|CIBER Enfermedades Cardiovasc, Madrid, Spain;
Yale Univ, Sch Med, Yale Cardiovasc Res Ctr, 333 Cedar St, New Haven, CT 06511 USA|Yale Univ, Sch Med, Yale Stem Cell Ctr, 333 Cedar St, New Haven, CT 06511 USA;
Yale Univ, Sch Med, Yale Cardiovasc Res Ctr, Vasc Biol & Therapeut Program, 333 Cedar St, New Haven, CT 06511 USA|Yale Univ, Sch Med, Dept Med, 333 Cedar St, New Haven, CT 06511 USA|Yale Univ, Sch Med, Dept Pharmacol, 333 Cedar St, New Haven, CT 06511 USA;
Boston Univ, Sch Med, Whitaker Cardiovasc Inst, Mol Cardiol, Boston, MA 02118 USA;
机译:DNMT3A和TET2中的功能性突变导致小鼠的动脉粥样硬化和收敛巨噬细胞表型加速
机译:随着动脉粥样硬化的增加,干细胞增殖加速克隆血液杂草
机译:补体调节蛋白CD59的缺乏加速了糖尿病诱导的小鼠动脉粥样硬化的发展
机译:动脉粥样硬化的实验模型的作用:Apoe-Nockout小鼠在制育新药物对动脉粥样硬化的作用
机译:缺乏Stat3-beta(全身性炎症的整体抑制因子)的小鼠的动脉粥样硬化加速。
机译:与TET2缺乏症相关的克隆性造血作用加速小鼠的动脉粥样硬化发展
机译:颜料上皮衍生因子缺乏通过促进用高脂血症的小鼠促进内皮脂肪酸摄取来加速动脉粥样硬化发育