机译:Dysbindin通过控制NMDA受体表面表达来调节海马LTP
Receptor Biology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892-3714 Medical Research Council Centre for Synaptic Plasticity, Department of Anatomy, University of Bristol, School of Medical Sciences, Bristol BS8 1TD, United Kingdom;
Section on Neural Development and Plasticity, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD 20892-3714;
Receptor Biology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892-3714;
Gene, Cognition and Psychosis Program, National Institute of Mental Health, National Institutes of Health, Bethesda, MD 20892-3714;
Section on Neural Development and Plasticity, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD 20892-3714;
Receptor Biology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892-3714;
Section on Neural Development and Plasticity, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD 20892-3714 Gene, Cognition and Psychosis Program, National Institute of Mental Health, National Institutes of Health, Bethesda, MD 20892-3714;
glutamate receptor; schizophrenia; synaptic plasticity;
机译:通过恐惧调节调节的海马NMDA GLUN2B受体的表面表达决定了其对成年大鼠内存整合的贡献
机译:活性钙/钙调蛋白依赖性蛋白激酶II(CAMKII)通过促进培养基中CAMKII和NMDA受体之间的相互作用来调节NMDA受体介导的出弱性长期增强(I-LTP)
机译:铜抑制NMDA受体依赖性LTP并调节小鼠海马切片中LTP后的配对脉冲比率
机译:模仿水凝胶的淋巴结刚度以分子亚型特异性方式调节人B细胞淋巴瘤的生长和细胞表面受体#的表达。
机译:通过调节mRNA衰减动力学来控制LDL受体的表达。
机译:Dysbindin通过控制NMDA受体表面表达来调节海马LTP
机译:Dysbindin通过控制NMDA受体表面表达来调节海马LTP