机译:通过消耗必需氨基酸和mTOR信号传导来抵抗感染
Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, United Kingdom;
Immunotherapy Center, Department of Medicine, Medical College of Georgia, Augusta, GA 30912;
Skirball Institute of Biomolecular Medicine, Department of Medicine, New York School of Medicine, New York, NY 10016;
Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, United Kingdom;
amino acid catabolism; foxp3; mTOR inhibitor; regulatory T cells; rapamycin;
机译:休息损害骨骼肌氨基酸转运蛋白表达,MTORCL信号传导和蛋白质合成,响应缩醛基本氨基酸
机译:AMP活化蛋白激酶(AMPK)信号和必需氨基酸对哺乳动物雷帕霉素靶标(mTOR)信号和乳腺细胞蛋白质合成速率的影响
机译:抵抗运动与摄入必需氨基酸相结合后单个人肌肉纤维中的mTORC1信号传导
机译:使用定制的拉曼光谱系统调查和校准非必需氨基酸
机译:摄入碳水化合物和支链氨基酸对响应于大量抵抗运动的胰岛素-PI3K-mTOR信号转导途径的骨骼肌蛋白质合成标记的影响。
机译:通过消耗必需氨基酸和mTOR信号传导来抵抗感染
机译:通过消耗必需氨基酸和mTOR信号转导来抵抗感染。