机译:竞争性反馈环在细胞微环境中塑造辅助和调节性T淋巴细胞之间的IL-2信号传导
Research Group Modeling of Biological Systems (B086), German Cancer Research Center, 69120 Heidelberg, Germany Theoretical Biophysics, Humboldt University, 10115 Berlin, Germany;
rnResearch Group Immunomodulation, German Rheumatology Research Center, 10117 Berlin, Germany;
rnTheoretical Biophysics, Humboldt University, 10115 Berlin, Germany;
rnTheoretical Biophysics, Humboldt University, 10115 Berlin, Germany;
rnResearch Group Modeling of Biological Systems (B086), German Cancer Research Center, 69120 Heidelberg, Germany;
rnResearch Group Immunomodulation, German Rheumatology Research Center, 10117 Berlin, Germany;
rnResearch Group Modeling of Biological Systems (B086), German Cancer Research Center, 69120 Heidelberg, Germany Theoretical Biophysics, Humboldt University, 10115 Berlin, Germany;
bi-stability; cell-to-cell communication; cytokine networks; mathematical modeling; reaction-diffusion systems;
机译:与治疗效应T淋巴细胞百分比的年龄相关差异及其在Helper T淋巴细胞中的健康个体和特征Satt1 / Stat5信号响应
机译:反馈回路在时空上塑造细胞信号
机译:通过调节性CD4 + CD25 + T淋巴细胞从幼稚CD4 + T细胞中的细胞周期进程中分离出IL-2信号。
机译:微环境和反馈信号回路在稳定疾病状态中的作用
机译:激活的T细胞中依赖细胞因子的Blimp-1表达通过负反馈调节回路抑制IL-2的产生。
机译:竞争性反馈环在细胞微环境中塑造辅助和调节性T淋巴细胞之间的IL-2信号传导
机译:竞争性反馈环在细胞微环境中塑造辅助和调节性T淋巴细胞之间的IL-2信号传导
机译:三种不同信号参与诱导人T淋巴细胞IL-2基因表达的证据。