机译:非规范性NF-kB调节诱导性共刺激(ICOS)配体表达和T滤泡辅助细胞发育
Department of Immunology, University of Texas MD Anderson Cancer Center, Houston, TX 77030;
Department of Immunology, University of Texas MD Anderson Cancer Center, Houston, TX 77030 Laboratory of Gene Regulation and Signal Transduction, Department of Pharmacology, School of Medicine, University of California San Diego, La Jolla, CA 92093;
Department of Immunology, University of Texas MD Anderson Cancer Center, Houston, TX 77030 Howard Hughes Medical Institute and Immunology Program, Memorial Sloan-Kettering Cancer Center, New York, NY 10065;
Department of Immunology, University of Texas MD Anderson Cancer Center, Houston, TX 77030;
Department of Immunology, University of Texas MD Anderson Cancer Center, Houston, TX 77030;
Department of Immunology, University of Texas MD Anderson Cancer Center, Houston, TX 77030 University of Texas Graduate School of Biomedical Sciences, Houston, TX 77030;
机译:共刺激分子ICOS在滤泡性T辅助细胞和TH-17细胞发育中调节c-Maf和IL-21的表达。
机译:白介素10缺乏症对树突状细胞的诱导型共刺激配体(ICOS-L)表达增强及其对呼吸道感染T细胞亚群的影响
机译:骨髓来源的浆细胞样树突状细胞的同种刺激活性独立于吲哚胺双加氧酶,但受诱导型共刺激配体表达的调节。
机译:通过抑制BV2微胶质细胞中的NF-KB活化,桦木酸抑制LPS诱导的MMP-9表达
机译:可诱导的共刺激物调节效应子CD4T细胞的运输和记忆CD4T细胞的维持。
机译:非规范性NF-κB调节诱导性共刺激(ICOS)配体表达和T滤泡辅助细胞的发育
机译:非规范性NF-κB调节诱导性共刺激(ICOS)配体表达和T滤泡辅助细胞的发育