机译:诱导型NOS诱导的氯化物细胞内通道4(CLIC4)核转运调节巨噬细胞的失活
Laboratory of Cancer Biology and Genetics, Center for Cancer Research, National Cancer Institute,Bethesda, MD 20892;
Laboratory of Cancer Biology and Genetics, Center for Cancer Research, National Cancer Institute,Bethesda, MD 20892;
Laboratory of Cancer Biology and Genetics, Center for Cancer Research, National Cancer Institute,Bethesda, MD 20892;
Laboratory of Cancer Biology and Genetics, Center for Cancer Research, National Cancer Institute,Bethesda, MD 20892;
Laboratory of Cancer Biology and Genetics, Center for Cancer Research, National Cancer Institute,Bethesda, MD 20892;
Laboratory of Cancer Biology and Genetics, Center for Cancer Research, National Cancer Institute,Bethesda, MD 20892;
Laboratory of Molecular Immunoregulation, Center for Cancer Research, National Cancer Institute,Bethesda, MD 20892;
Laboratory of Cancer Biology and Genetics, Center for Cancer Research, National Cancer Institute,Bethesda, MD 20892;
protein nitrosylation; il-1beta; phagocytosis;
机译:S-亚硝基化调节氯化物细胞内通道蛋白CLIC4的核易位
机译:氯化物细胞内通道蛋白CLIC4抑制由Formin MDIA2的组成型活性突变体诱导的氟化钠形成
机译:细胞内氯通道蛋白CLIC1和CLIC4诱导IL-1β转录并激活NLRP3炎性小体
机译:mtCLIC / CLIC4,一种氯通道蛋白,参与细胞凋亡并定位于线粒体的内膜
机译:N型钙通道的细胞内结构域调节失活并结合新型分泌蛋白
机译:诱导型NOS诱导的氯化物细胞内通道4(CLIC4)核转运调节巨噬细胞的失活
机译:S-亚硝基化调节氯化物细胞内通道蛋白CLIC4 *的核转运
机译:巨噬细胞活化杀死利什曼原虫:重组干扰素-γ和非干扰素淋巴因子均可诱导巨噬细胞对细胞内破坏无鞭毛体的活化