机译:用Pin1在胶质瘤干细胞中促进的Sumo1促进蛋白质Sumoglation扩大胶质母细胞瘤恶性肿瘤
Cleveland Clin Dept Canc Biol Lerner Res Inst 9500 Euclid Ave NE30 Cleveland OH 44195 USA;
Cleveland Clin Dept Canc Biol Lerner Res Inst 9500 Euclid Ave NE30 Cleveland OH 44195 USA;
Cleveland Clin Dept Canc Biol Lerner Res Inst 9500 Euclid Ave NE30 Cleveland OH 44195 USA;
Cleveland Clin Dept Canc Biol Lerner Res Inst 9500 Euclid Ave NE30 Cleveland OH 44195 USA;
Cleveland Clin Dept Canc Biol Lerner Res Inst 9500 Euclid Ave NE30 Cleveland OH 44195 USA;
Cleveland Clin Dept Canc Biol Lerner Res Inst 9500 Euclid Ave NE30 Cleveland OH 44195 USA|Cleveland Clin Dept Radiat Oncol Cleveland OH 44195 USA|Case Western Reserve Univ Sch Med Case Comprehens Canc Ctr Cleveland OH USA;
Case Western Reserve Univ Sch Med Case Comprehens Canc Ctr Cleveland OH USA|Case Western Reserve Univ Univ Hosp Brain Tumor & Neuro Oncol Ctr Cleveland OH USA;
Univ Calif San Diego Dept Med Div Regenerat Med San Diego CA 92103 USA;
Univ Sci & Technol China Affiliated Hosp USTC 1 Intelligent Pathol Inst Div Life Sci & Med Hefei 230001 Anhui Peoples R China;
Cleveland Clin Dept Canc Biol Lerner Res Inst 9500 Euclid Ave NE30 Cleveland OH 44195 USA|Case Western Reserve Univ Univ Hosp Brain Tumor & Neuro Oncol Ctr Cleveland OH USA|Cleveland Clin Lerner Res Inst Ctr Canc Stem Cell Res Cleveland OH 44195 USA;
glioma stem cells; Pin1; PML; posttranslational modification; protein sumoylation;
机译:CentroSomal蛋白55的过度表达调节胶质瘤细胞的增殖,并在Glioblastoma U251细胞中促进EGFRVIII的介导的增殖
机译:缺氧诱导的USP22-BMI1轴通过调节HIF-1α来促进胶质瘤干细胞的茎秆和恶性肿瘤
机译:腺病毒感染通过TLR9 / Neat1 / Stat3途径促进从胶质母细胞瘤细胞形成胶质瘤干细胞
机译:骨形态发生蛋白调节人胶质母细胞瘤干细胞中的致瘤性
机译:HIF1a介导的STAT3激活促进自发性鼠神经胶质瘤衍生的干样细胞的自我更新和恶性肿瘤。
机译:照射小儿胶质母细胞瘤细胞可通过全基因组转录组变化促进放射抵抗并增强神经胶质瘤恶性程度
机译:儿科胶质母细胞瘤细胞的照射促进辐射敏感度并通过基因组的转录组变化提高胶质瘤恶性肿瘤