机译:视网膜母细胞瘤中p53途径的失活
St Jude Childrens Hosp, Dept Dev Neurobiol, Memphis, TN 38105 USA;
St Jude Childrens Hosp, Dept Chem Biol & Therapeut, Memphis, TN 38105 USA;
St Jude Childrens Hosp, Dept Pathol, Memphis, TN 38105 USA;
St Jude Childrens Hosp, Dept Surg, Div Ophthalmol, Memphis, TN 38105 USA;
St Jude Childrens Hosp, Dept Hematol Oncol, Memphis, TN 38105 USA;
Univ Calif San Francisco, Dept Mol & Cellular Pharmacol, San Francisco, CA 94143 USA;
Leiden Univ, Med Ctr, Dept Mol & Cell Biol, NL-2300 RC Leiden, Netherlands;
Univ Tennessee, Ctr Hlth Sci, Dept Ophthalmol, Memphis, TN 38163 USA;
FIRC Inst Mol Oncol, I-20139 Milan, Italy;
Flanders Interuniv Biotechnol, Lab Mol Canc Biol, B-9052 Ghent, Belgium;
TUMOR-SUPPRESSOR; RETINAL DEVELOPMENT; MDM2; CELL; CANCER; RB; DEATH; AMPLIFICATION; ACTIVATION; INHIBITORS;
机译:视网膜母细胞瘤和Trp53肿瘤抑制物的体细胞失活诱导的自发性鳞状细胞癌。
机译:PDCD2的条件失活诱导p53活化和细胞周期阻滞PDCD2的条件失活诱导p53活化和细胞周期阻滞PDCD2的条件失活诱导p53活化和细胞周期阻滞
机译:人类T细胞淋巴病毒1型税灭活p53需要激活NF-κB途径,并依赖于p53磷酸化
机译:通过灭活视网膜母细胞瘤敏感性基因和P53的小鼠肿瘤模型:当前状态和未来方向
机译:人巨细胞病毒编码的激酶UL97对视网膜母细胞瘤家族蛋白的磷酸化和失活
机译:人类T细胞淋巴病毒1型税灭活p53需要激活NF-κB途径并依赖于p53磷酸化
机译:由视网膜母细胞瘤和Trp53肿瘤抑制因子的体细胞失活诱导的自发性鳞状细胞癌。癌症研究