机译:线粒体蛋白质失衡作为长寿机制
Laboratory for Integrative and Systems Physiology, Ecole Polytechnique Federale de Lausanne,CH-1015 Lausanne,Switzerland,Laboratory Genetic Metabolic Diseases, Academic Medical Center,1105 AZ Amsterdam, The Netherlands;
Laboratory for Integrative and Systems Physiology, Ecole Polytechnique Federale de Lausanne,CH-1015 Lausanne,Switzerland;
Laboratory for Integrative and Systems Physiology, Ecole Polytechnique Federale de Lausanne,CH-1015 Lausanne,Switzerland;
Laboratory for Integrative and Systems Physiology, Ecole Polytechnique Federale de Lausanne,CH-1015 Lausanne,Switzerland;
Laboratory for Integrative and Systems Physiology, Ecole Polytechnique Federale de Lausanne,CH-1015 Lausanne,Switzerland;
BioEM Facility, Ecole Polytechnique Federale de Lausanne, CH-1015 Lausanne, Switzerland;
Department of Anatomy and Neurobiology and Center for Integrative and Translational Genomics, Memphis, Tennessee 38163, USA;
Laboratory for Integrative and Systems Physiology, Ecole Polytechnique Federale de Lausanne,CH-1015 Lausanne,Switzerland;
机译:高温需求蛋白A2蛋白酶活性的损失通过SARCEPENIA中的线粒体生物血栓生物发生的差异调节诱导QITON核心不平衡
机译:Beta alpha重复蛋白的拓扑受挫:序列多样性调节了alpha / beta / alpha夹心蛋白的保守折叠机制。
机译:IFTA-2是一种保守的纤毛蛋白,参与调节秀丽隐杆线虫的寿命和dauer形成的途径
机译:利用新兴的基因组数据确定保守的骨形态发生蛋白(Bmp)2基因表达机制
机译:保守的长寿干预的遗传学和分子机制的探索。
机译:线粒体蛋白质失衡作为长寿机制
机译:线粒体蛋白质失衡作为长寿机制