机译:肿瘤坏死因子α抑制2型碘甲状腺素脱碘酶的新机制:蛋白酶体降解
Department of Clinical Laboratory Medicine, Gunma University Graduate School of Medicine, Maebashi 371-8511, Japan;
Department of Clinical Laboratory Medicine, Gunma University Graduate School of Medicine, Maebashi 371-8511, Japan;
Department of Clinical Laboratory Medicine, Gunma University Graduate School of Medicine, Maebashi 371-8511, Japan;
Department of Clinical Laboratory Medicine, Gunma University Graduate School of Medicine, Maebashi 371-8511, Japan;
Department of Medicine and Molecular Sciences, Gunma University Graduate School of Medicine, Maebashi 371-8511, Japan;
Department of Clinical Laboratory Medicine, Gunma University Graduate School of Medicine, Maebashi 371-8511, Japan;
Nonthyroidal illness; Type 2 iodothyronine deiodinase; Tumor necrosis factor α; Proteasome;
机译:肿瘤坏死因子α抑制2型碘甲状腺素脱碘酶的新机制:蛋白酶体降解的参与。
机译:马齿By叶的丁醇部分的抗炎作用:体内实验涉及抑制肿瘤坏死因子α,前列腺素E-2产生和多形核白细胞迁移的机制
机译:涉及ERK的新机制有助于罗格列酮抑制人内皮细胞中的肿瘤坏死因子-α和干扰素-γ的炎症作用。
机译:抑制细胞内特例化诱导淀粉样蛋白促甲蛋白变体的蛋白酶体降解
机译:肿瘤坏死因子受体相关因子3(TRAF3)在调节I型和II型NF-kappaB途径中的作用。
机译:肿瘤坏死因子-α和干扰素-γ通过转化生长因子-β1抑制人类I型胶原基因表达的激活。在转录和转录后水平存在两种不同抑制机制的证据。
机译:肿瘤坏死因子α抑制2型碘硫酸滴水酶的新机制:蛋白酶体降解的累积