机译：核PGC1α蛋白的补偿性增加主要与ZDF大鼠的肌膜下线粒体适应有关。
Graham P. Holloway,1 Brendon J. Gurd,2 Laelie A. Snook,1 Jamie Lally,1 and Arend Bonen1From the 1 Department of Human Health and Nutritional Sciences, University of Guelph, Guelph, Ontario, Canada, and the -School of Kinesiology, Queen's University, Kingston, Ontario, CanadaCorresponding author: Graham Holloway, ghollowa@uoguelph.ca.Received 13 October 2009 and accepted 17 January 2010. Published ahead of print at http://diabetes.diabetesjoiumals.org on 26 January 2010. DOI: 10.2337/db09-1519.G. P.H. and B.J.G. contributed equally to this manuscript.© 2010 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.The costs of publication of this article were defrayed in part by the payment of page charges. This article must therefore be hereby marked "advertisement" in accordarne with 18 U.S.C. Section 1734 solely to indicate this fact.,;
机译：过氧化物酶体增殖物激活受体α（PPARα）激活剂，苯扎贝特和Wy-14,643，在不改变大鼠前脂肪细胞原代培养的线粒体膜电位的情况下，增加了解偶联蛋白3 mRNA水平。
机译：收缩诱导原代大鼠骨骼肌细胞中过氧化物酶体增殖物激活受体（PPAR）-γ共激活因子1 alpha（PGC-1 alpha），线粒体解偶联蛋白3（UCP3）和己糖激酶II（HKII）基因表达的增加。
机译：甲状腺素对大鼠肝核甲状腺激素受体的作用。甲状腺素与核非组蛋白的结合及线粒体α-甘油磷酸脱氢酶活性的诱导
机译：子系统肠道线粒体蛋白的相对定量
机译：卵巢激素通过对脑血管中PGC1亚型的不同调节来提高线粒体效率和生物发生。
机译：核PGC1α蛋白的补偿性增加主要与ZDF大鼠肌膜下线粒体适应有关。
机译：核PGC1α蛋白的补偿性增加主要与ZDF大鼠肌膜下线粒体适应有关。
