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Inducible Nitric Oxide Synthase Induction Underlies Lipid-Induced Hepatic Insulin Resistance in Mice: Potential Role of Tyrosine Nitration of Insulin Signaling Proteins

机译:诱导型一氧化氮合酶诱导基础的小鼠脂质诱导的肝胰岛素抵抗:胰岛素信号蛋白酪氨酸硝化的潜在作用。

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摘要

The present study was undertaken to assess the contribution of inducible nitric oxide (NO) synthase (iNOS) to lipid-induced insulin resistance in vivo.
机译:进行本研究以评估诱导型一氧化氮(NO)合酶(iNOS)对体内脂质诱导的胰岛素抵抗的作用。

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  • 来源
    《Diabetes》 |2010年第4期|p.861-871|共11页
  • 作者单位

    From the Axe Cardiologie, Centre de Recherche de l'Institut Universitaire de Cardiologie et de Pneumologie de Québec, Québec, Canada, and Centre Hospitalier Universitaire de Québec, Axe Métabolisme, Santé Vasculaire et Rénale, Department of Medicine, Laval University, Québec, Canada.Corresponding author: André Marette, andre.marette@crchul.ulaval.ca.Received 19 August 2009 and accepted 17 January 2010. Published ahead of print at htrp://diabetes.diabetesjournals.org on 26 January 2010. DOI: 10.2337/db09-1238.© 2010 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit. and the work is not altered. See http://creativecommons.org/licenses/l^y -nc-nd/3.0/ for details.The costs of publication of this article were defrayed in part by the payment of page rharges. This article must therefore be hereby marked "advertisement" in accordance with 18 U.S.C. Section 1734 solely to indicate this fart.;

  • 收录信息 美国《科学引文索引》(SCI);美国《化学文摘》(CA);
  • 原文格式 PDF
  • 正文语种 eng
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