机译:体力活动导致线粒体生物发生和过氧化物酶体增殖物激活的受体-γ共激活因子-1α(PGC-1α)脱乙酰化:需要完整的脂肪细胞因子信号传导
Ling Li,1 Ruping Pan,1 Rong Li,' Bernd Niemann,2 Anne-CathJeen Aurich,1 Ying Chen,1 and Susanne Rohrbach1,3From the 'Institute of Physiology, Justus Liebig University Giessen, Giessen, Germany, the ^Department of Cardiac and Vascular Surgery, Justus Liebig University Giessen, Giessen, Germany, and the :,mstitute of Pathophysiology, Martin Luther University Halle-Wittenberg, Halle, Germany.Corresponding author Susanne Rohrbach, susanne.rohrbach@physiologie. med.uni-giesseri.de.Received 10 March 2010 and accepted 1 October 2010. Published ahead of print at http://diabetes.diabetesjournals.org on 7 October 2010. DOI: 10.2337/dbl0-0a31.L.L. and R.P, contributed equally to this work.© 2011 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/Iicenses/by -nc-nd/3.0/ for details.The costs of publication of this article were defrayed in part by the payment of page charges. This article must therefore be hereby marked "advertiseinent" in accordance with 18 U.S.C. Section 1734 solely to Indicate this fact.,;
机译:线粒体生物发生或过氧化物体增殖物激活的受体-γ共同诱导剂-1α(PGC-1α)脱乙酰化后不需要脱桂顿1(SIRT1)脱乙酰酶活性。耐久性运动
机译:线粒体生物发生和过氧化物酶体增殖物激活受体7共激活因子-la(PGC-la)脱乙酰化的体育活动完整的脂肪细胞因子信号是必需的
机译:过氧化物酶体增殖物激活受体γcoactivator-1 alpha(PGC-1 alpha)的线粒体转录因子A(Tfam)启动子的Gly(482)变体的coactivator活性受损
机译:甲状腺激素对丙酮酸脱氢酶激酶4的调节:过氧化物酶体增殖物激活受体γcoactivator-1 alpha和CCAAT增强剂结合蛋白的作用
机译:耐力运动后线粒体生物发生或过氧化物酶体增殖物激活的受体-γCoactivator-1α(PGC-1α)脱乙酰化不需要Sirtuin 1(SIRT1)脱乙酰酶活性
机译:身体活动导致线粒体生物发生和过氧化物酶体增殖物激活的受体-γ共激活因子-1α(PGC-1α)脱乙酰化:需要完整的脂肪细胞因子信号传导