机译:血红素调节的elF2α激酶调节肝FGF21,并被PPARβ/δ缺乏激活。
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain,CIBERDEM, Instituto de Salud Carlos Ⅲ, Madrid, Spain,Pediatric Research Institute, Hospital Sant Joan de Deu, Barcelona, Spain;
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain,CIBERDEM, Instituto de Salud Carlos Ⅲ, Madrid, Spain,Pediatric Research Institute, Hospital Sant Joan de Deu, Barcelona, Spain;
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain;
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain;
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain;
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain;
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain;
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain,CIBERDEM, Instituto de Salud Carlos Ⅲ, Madrid, Spain,Pediatric Research Institute, Hospital Sant Joan de Deu, Barcelona, Spain;
CIBERDEM, Instituto de Salud Carlos Ⅲ, Madrid, Spain,Instituto de Investigaciones Biomedicas Alberto Sols (CSIC/UAM), Madrid, Spain;
CIBERDEM, Instituto de Salud Carlos Ⅲ, Madrid, Spain,Instituto de Investigaciones Biomedicas Alberto Sols (CSIC/UAM), Madrid, Spain;
CIBERDEM, Instituto de Salud Carlos Ⅲ, Madrid, Spain,Instituto de Investigaciones Biomedicas Alberto Sols (CSIC/UAM), Madrid, Spain;
Pediatric Research Institute, Hospital Sant Joan de Deu, Barcelona, Spain,Department of Biochemistry and Molecular Biology and Institute of Biomedicine of the University of Barcelona, Barcelona Spain,CIBEROBN, Instituto de Salud Carlos Ⅲ, Madrid, Spain;
Pediatric Research Institute, Hospital Sant Joan de Deu, Barcelona, Spain,Department of Biochemistry and Molecular Biology and Institute of Biomedicine of the University of Barcelona, Barcelona Spain,CIBEROBN, Instituto de Salud Carlos Ⅲ, Madrid, Spain;
Center for Integrate Genomics, University of Lausanne, Lausanne, Switzerland,Lee Kong Chian School of Medicine, Nanyang Technological University, Singapore, Singapore,INRA ToxAlim, UMR1331, Chemin de Toumefeuille, Toulouse Cedex, France;
Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Institute of Biomedicine of the University of Barcelona, Barcelona, Spain,CIBERDEM, Instituto de Salud Carlos Ⅲ, Madrid, Spain,Pediatric Research Institute, Hospital Sant Joan de Deu, Barcelona, Spain;
机译:血红素缺乏症大鼠肝细胞中地塞米松介导的色氨酸2,3-双加氧酶受损的诱导:肝elF2α的翻译控制:激酶,血红素调节的抑制剂11
机译:脂肪酸去饱和酶1通过调节PPARα-FGF21轴来影响肝脂质稳态稳态
机译:PPARβ/δ和FGF21对VLDL受体的肝调节作用可调节非酒精性脂肪肝
机译:FGF21通过PPAR-
机译:调节肝载脂蛋白B100产生的细胞内信号传导途径:促分裂原活化蛋白激酶(MAPK)和NFkappBB激酶(IKK)-NFkappaB抑制剂的作用。
机译:PPARβ/δ和FGF21对VLDL受体的肝调节作用可调节非酒精性脂肪肝
机译:血红素调节的eIF2α激酶调节肝FGF21并被PPARβ/δ缺乏激活