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首页> 外文期刊>Japanese heart journal >Heart Sounds in Bundle Branch BlockObservations on 244 Cases
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Heart Sounds in Bundle Branch BlockObservations on 244 Cases

机译:244例束支传导阻滞观察中的心音

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The heart sounds in 244 cases of bundle branch block observed during the years 1926-1963 were measured in the quantitative symbol phonocardiograms and electrophonocardiograms. Splitting of the first sound occurs rarely in both right and left bundle branch block and is most likely to be detected at the apex. In the QSPcg's. splitting of the second sound occurred in about 40% of Series A (1926-1952) and 80% of Series B (1952-1963) at the left border of the sternum near the fourth inter costal space with the patient in the sitting position. In Series B, auscultation with the patient in the dorsal recumbent posture, splitting was detected in only 60% at this area. The tables depict the frequency of splitting at all areas of auscultation. The left ventricular components of both the first and second sounds are shorter than normal. A first sound of markedly reduced loudness occurs with somewhat increased frequency in left bundle branch block. The range of loudness of the first sound is otherwise normal in both types. The right ventricular component (tricuspid and pulmonic) is a short thud which is rather faint in about twothirds of cases and about half the loudness of a normal first sound in the remaining third. The interval of quiet between the 2 components does not vary with respiration but the loudness of the pulmonic component increases during inspiration and decreases during expiration, sometimes to the point of extinction. Left bundle branch block is strongly suggested when the pulmonic component precedes the aortic. A rare exception occurs when pulmonary arterial pressure is high enough to make the pulmonic component in the case of right bundle branch block louder than the aortic which precedes it. In about a third of cases of right bundle branch block, physiologic splitting of the first sound could be detected which indicates that this type of split does not depend upon asynchronism of the ventricles. In about 20% of cases with satisfactory electrophonocardiograms, splitting was absent although it was detected by clinical auscultation in the sitting postion. This probably results from the fact that during clinical auscultation the pulmonic component of the second sound became inaudible in about 30% of cases when the position was changed from sitting to dorsal recumbent. The electrophonocardiograms were recorded in this latter posture. The absence of splitting in any posture in some instances is due to the faintness of the right ventricular components of the first and second sounds. In others, it may be due to the fact that the electrocardiogram may present the pattern of bundle branch block but the mechanical events may not be correspondingly asynchronous.
机译:用定量符号心电图和心电图测量了1926-1963年间观察到的244例束支传导阻滞的心音。第一声​​音的分裂很少在左右束支传导阻滞中发生,并且最有可能在顶点处被检测到。在QSPcg中。当患者处于坐姿时,在第四个肋间隙附近的胸骨左边界,第二种声音的分裂发生在大约40%的A系列(1926-1952)和B系列(1952-1963)中。在系列B中,以患者的仰卧姿势进行听诊,在该区域仅发现60%的分裂。这些表描述了在所有听诊区域的分裂频率。第一声​​和第二声的左心室分量都比正常人短。在左束支传导阻滞中出现频率显着增加的响度明显降低的第一声音。否则,两种声音中第一声音的响度范围都是正常的。右心室成分(三尖瓣和肺动脉)是一短声,在大约三分之二的情况下是微弱的,而在其余的三分之一中则是正常的第一声音的一半。这两个成分之间的安静时间间隔不会随呼吸而变化,但肺部声音的响度在吸气时会增加,而在呼气时会降低,有时甚至会消失。当肺动脉组件先于主动脉时,强烈建议左束支传导阻滞。当肺动脉压高到足以使右束支传导阻滞的肺动脉组件比其前的主动脉大时,会发生罕见的例外。在右束支传导阻滞的大约三分之一情况下,可以检测到第一声音的生理分裂,这表明这种分裂不取决于心室的异步性。在心电图满意的情况下,约有20%的患者没有分裂,尽管通过坐位的临床听诊可以发现分裂。这可能是由于以下事实:在临床听诊期间,当位置从坐着改为背卧时,第二声音的肺音成分在大约30%的情况下听不到。心电图以后一种姿势记录。在某些情况下,没有任何姿势上的分裂是由于第一声音和第二声音的右心室成分微弱。在其他情况下,可能是由于心电图可能呈现束支传导阻滞的模式,但机械事件可能并不相应地异步。

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