Activation in M1 but not M2 Macrophages Contributes to Cardiac Remodeling after Myocardial Infarction in Rats: a Critical Role of the Calcium Sensing Receptor/NRLP3 Inflammasome

Wenxiu Liu; Xin Zhang; Meng Zhao; Xiaohui Zhang; Jinyu Chi; Yue Liu; Fang Lin; Yu Fu; Dandan Ma; Xinhua Yin

首页> 外文期刊>Cellular Physiology and Biochemistry >Activation in M1 but not M2 Macrophages Contributes to Cardiac Remodeling after Myocardial Infarction in Rats: a Critical Role of the Calcium Sensing Receptor/NRLP3 Inflammasome

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Activation in M1 but not M2 Macrophages Contributes to Cardiac Remodeling after Myocardial Infarction in Rats: a Critical Role of the Calcium Sensing Receptor/NRLP3 Inflammasome

机译：M1而不是M2巨噬细胞的激活有助于大鼠心肌梗死后的心脏重塑：钙敏感受体/ NRLP3炎性体的关键作用。

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《Cellular Physiology and Biochemistry》 |2015年第6期|共18页
作者
Wenxiu Liu; Xin Zhang; Meng Zhao; Xiaohui Zhang; Jinyu Chi; Yue Liu; Fang Lin; Yu Fu; Dandan Ma; Xinhua Yin;
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中图分类细胞生物物理学;
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1. IL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation [J] . Jung Mira, Ma Yonggang, Iyer Rugmani Padmanabhan, Basic Research in Cardiology: Official Journal of the German Association of Cardiovascular Research . 2017,第3期

机译：通过刺激M2巨噬细胞极化和成纤维细胞活化，IL-10通过心肌梗死后改善心脏重塑
2. Macrophages from NLRP3 and Caspase-1 null mice exhibit reduced inflammasome activation, altered expression of M1/M2 markers and oxidative stress, ER stress and autophagy genes in response to iron: Role for NLRP3 inflammasome in iron-mediated liver injury. [J] . Handa Priya, Morgan-Stevenson Vicki, Sekine Aimee, Hepatology: Official Journal of the American Association for the Study of Liver Diseases . 2017,第Suppla1期

机译：来自NLRP3和Caspase-1核小鼠的巨噬细胞表现出降低的炎症组活化，改变M1 / M2标记和氧化应激，ER应激和自噬基因的表达，响应于Iron：在铁介导的肝损伤中的NLRP3炎性作用。
3. IL-6 knockout ameliorates myocardial remodeling after myocardial infarction by regulating activation of M2 macrophages and fibroblast cells [J] . R. Jing, T.-Y. Long, W. Pan, European review for medical and pharmacological sciences. . 2019,第14期

机译：IL-6敲除可通过调节M2巨噬细胞和成纤维细胞的活化来改善心肌梗塞后的心肌重塑
4. Mathematical modeling of macrophage activation in left ventricular remodeling post-myocardial infarction [C] . Wang Yunji, Jin Yufang, Ma Yonggang, Genomic Signal Processing and Statistics (GENSIPS), 2011 IEEE International Workshop on . 2011

机译：心肌梗死后左心室重构中巨噬细胞激活的数学模型
5. The effects of resistance versus endurance exercise on cardiac function, myocardial remodeling and hormone response in post-myocardial infarction rats. [D] . Wason, Emily A. 2016

机译：抵抗力与耐力运动对心肌梗死后大鼠心功能，心肌重塑和激素反应的影响。
6. IL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation [O] . Mira Jung, Yonggang Ma, Rugmani Padmanabhan Iyer, -1

机译：IL-10通过刺激M2巨噬细胞极化和成纤维细胞活化来改善心肌梗死后的心脏重塑
7. Activation in M1 but not M2 Macrophages Contributes to Cardiac Remodeling after Myocardial Infarction in Rats: a Critical Role of the Calcium Sensing Receptor/NRLP3 Inflammasome [O] . Wenxiu Liu, Xin Zhang, Meng Zhao, 2015

机译：m1中的活化而不是m2巨噬细胞有助于大鼠心肌梗死后的心脏重塑：钙传感受体/ NRLp3炎症小体的关键作用

Activation in M1 but not M2 Macrophages Contributes to Cardiac Remodeling after Myocardial Infarction in Rats: a Critical Role of the Calcium Sensing Receptor/NRLP3 Inflammasome

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