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机译:重症MuSK重症肌无力B细胞免疫失调和自身抗体产生的潜在机制
Department of Neurology,Yale School of Medicine,New Haven,Connecticut;
Department of Neurology,Yale School of Medicine,New Haven,Connecticut;
Department of Neurology,Yale School of Medicine,New Haven,Connecticut;
Neurology Department,Hospital de la Santa Creu i Sant Pau,Barcelona,Spain;
Department of Neurology,Yale School of Medicine,New Haven,Connecticut;
Department of Neurology,Yale School of Medicine,New Haven,Connecticut;
myasthenia gravis; B cells; autoimmunity; immunopathology; autoantibodies; MuSK;
机译:在兔重症肌无力模型中,二价和单价自身抗体通过不同的机制导致MuSK功能障碍。
机译:二价和单价自身抗体通过兔子模型的肌肌无集模型引起麝香的功能障碍
机译:HLA-DRB1 HLA-DRB1至IgG4自身抗体和细胞因子生成的关系在肌肉特异性酪氨酸激酶Myasthenia Gravis(Musk-Mg)中
机译:麝香Myasthenia的致病性IgG4亚类自身抗体
机译:肌肉特异性激酶(MUSK)引起的重症肌无力的小鼠模型,直接蛋白相互作用调节大电导(BK)钙激活钾通道的门控和转运
机译:MuSK MG中B细胞免疫失调和自身抗体产生的潜在机制
机译:B细胞免疫诱导和自身抗体产量的机制肌肉肌炎肌炎