机译:C5a或补体膜攻击复合物协同增强趋化因子生成和肺损伤。
From the Department of Trauma Surgery,University of Freiburg Medical School, Freiburg/Breisgau, Germany|and the Department of Pathology,University of Michigan Medical School, Ann Arbor, Michigan;
the Department of Surgery,University of Louisville School of Medicine, Louisville, Kentucky|and the Department of Pathology,University of Michigan Medical School, Ann Arbor, Michigan;
From the Department of Trauma Surgery,University of Freiburg Medical School, Freiburg/Breisgau, Germany;
From the Department of Trauma Surgery,University of Freiburg Medical School, Freiburg/Breisgau, Germany;
From the Department of Trauma Surgery,University of Freiburg Medical School, Freiburg/Breisgau, Germany;
and the Department of Pathology,University of Michigan Medical School, Ann Arbor, Michigan;
机译:IgG免疫复合物诱导的皮肤和肺损伤中肥大细胞和补体成分C3 / C5a受体的组织特定部位要求
机译:IgG免疫复合物诱导的皮肤和肺损伤中肥大细胞和补体成分C3 / C5a受体的组织特定部位要求
机译:抑制补体系统的膜攻击复合物可减少小鼠颅脑外伤后继发性神经轴突丢失并促进神经系统恢复。
机译:补体激活产物和受体在肺炎性损伤中的作用。
机译:趋化因子和补体:了解它们在新生儿脑损伤和脑发育中的作用。
机译:C5a或补体膜攻击复合物协同增强趋化因子生成和肺损伤。
机译:C5a或补体膜攻击复合物协同增强趋化因子生成和肺损伤。