机译:C-C趋化因子受体2(CCR2)基因敲除对2型(血吸虫抗原消除)肺肉芽肿形成的影响:细胞募集和细胞因子反应的分析。
From the Departments of Pathology and Laboratory Medicine,Veterans Affairs Medical Center, Ann Arbor, Michigan;
and the Gladstone Institute of Cardiovascular Disease,University of California, San Francisco, California;
From the Departments of Pathology and Laboratory Medicine,Veterans Affairs Medical Center, Ann Arbor, Michigan;
and Pulmonary Medicine,Veterans Affairs Medical Center, Ann Arbor, Michigan;
the Department of Pathology,University of Michigan Hospitals, Ann Arbor, Michigan;
and Pulmonary Medicine,Veterans Affairs Medical Center, Ann Arbor, Michigan;
the Department of Pathology,University of Michigan Hospitals, Ann Arbor, Michigan;
and the Gladstone Institute of Cardiovascular Disease,University of California, San Francisco, California;
From the Departments of Pathology and Laboratory Medicine,Veterans Affairs Medical Center, Ann Arbor, Michigan|the Department of Pathology,University of Michigan Hospitals, Ann Arbor, Michigan;
机译:白细胞介素4和13参与分枝杆菌(1型)和血吸虫(2型)抗原引起的肺肉芽肿形成:细胞募集,趋化因子表达和细胞因子网络的多参数分析。
机译:分支杆菌(1型)和血吸虫(2型)抗原引起的肺肉芽肿形成中的趋化因子表达动力学。
机译:AMD3465,一种新型的CXCR4受体拮抗剂,可消除血吸虫抗原诱导的(2型)肺肉芽肿的形成。
机译:人类牙周受体人口响应中信息含量的神经网络分析
机译:分析锌(环境巴尔的摩细颗粒物的主要成分)在体外引起细胞因子和趋化因子释放并破坏细胞紧密连接中的作用。
机译:C-C趋化因子受体2(CCR2)基因敲除对2型(血吸虫抗原清除)肺肉芽肿形成的影响。
机译:C-C趋化因子受体2(CCR2)敲除对2型(血吸体抗原引出)肺肉芽肿形成的影响