机译:肺上皮细胞中Fas而非Caspase-8沉默可改善失血性休克和败血症后的肺细胞凋亡,炎症和中性粒细胞流入
From the Shock-Trauma Research Laboratories,Rhode Island Hospital and Brown University School of Medicine, Providence;
From the Shock-Trauma Research Laboratories,Rhode Island Hospital and Brown University School of Medicine, Providence;
and the Department of Cell and Molecular Biology,University of Rhode Island, Kingston, Rhode Island;
From the Shock-Trauma Research Laboratories,Rhode Island Hospital and Brown University School of Medicine, Providence;
Division of Surgical Research, and the Department of Surgery,Rhode Island Hospital and Brown University School of Medicine, Providence;
Division of Surgical Research, and the Department of Surgery,Rhode Island Hospital and Brown University School of Medicine, Providence;
From the Shock-Trauma Research Laboratories,Rhode Island Hospital and Brown University School of Medicine, Providence;
机译:IFN-γ在A549肺上皮细胞中增强FAS和轨迹介导的凋亡:通过干扰素应答元件增强Caspase-8表达。
机译:失血性休克引发的肺细胞因子诱导的中性粒细胞趋化因子表达增加:脂多糖引起的肺部炎症。
机译:出血性休克素在肺中由细胞因子诱导的中性粒细胞趋化因子表达增加表达:脂多糖后肺炎症中的作用
机译:肺上皮细胞中stat3通路的过度激活引起肺部炎症和肺癌
机译:失血性休克和吸入氧气的分数对大鼠肺和diaphragm肌中过氧化氢和细胞凋亡的影响。
机译:肺上皮细胞中Fas而非Caspase-8沉默可改善失血性休克和败血症后的肺细胞凋亡炎症和中性粒细胞流入
机译:肺上皮细胞中Fas而非Caspase-8沉默可改善失血性休克和败血症后的肺细胞凋亡,炎症和中性粒细胞流入
机译:灵长类动物失血性休克:肺结构和功能。治疗和未治疗失血性休克的肺部变化。治疗失血性休克的肺部变化。