Endothelial-Mesenchymal Transition as a Novel Mechanism for Generating Myofibroblasts during Diabetic Nephropathy

Akane Kizu; Damian Medici; Raghu Kalluri

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Endothelial-Mesenchymal Transition as a Novel Mechanism for Generating Myofibroblasts during Diabetic Nephropathy

机译：内皮-间充质转化作为糖尿病肾病过程中生成成纤维细胞的新机制

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This Commentary provides perspective on epithelial-mesenchymal transition in diabetic nephropathy.

机译：该评论为糖尿病性肾病中上皮间质的转变提供了视角。

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来源
《American Journal of Pathology》 |2009年第4期|1371-1373|共3页
作者
Akane Kizu; Damian Medici; Raghu Kalluri;
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作者单位

From the Division of Matrix Biology,Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston;

From the Division of Matrix Biology,Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston;

From the Division of Matrix Biology,Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston|the Department of Biological Chemistry and Molecular Pharmacology,Harvard Medical School, Boston|and the Harvard-Massachusetts Institute of Technology Division of Health Sciences and Technology,Boston, Massachusetts;

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1. The implication of Numb-induced Notch signaling in endothelial-mesenchymal transition of diabetic nephropathy [J] . Wei Liu, Yanhua Wu, Feng Yu, Journal of diabetes and its complications . 2018,第10期

机译：Numb诱导的Notch信号传导在糖尿病肾病的内皮 - 间充质转换中的含义
2. Serum response factor induces endothelial-mesenchymal transition in glomerular endothelial cells to aggravate proteinuria in diabetic nephropathy [J] . Zhao Long, Zhao Jun, Wang Xueling, Physiological genomics . 2016,第10期

机译：血清反应因子诱导糖尿病肾病肾小球内皮细胞内皮-间质转化，加重蛋白尿
3. Blockade of Endothelial-Mesenchymal Transition by a Smad3 Inhibitor Delays the Early Development of Streptozotocin-Induced Diabetic Nephropathy [J] . Jinhua Li Xinli Qu Jun Yao Georgina Caruana Sharon D Ricardo Yasuhiko Yamamoto Hiroshi Yamamoto John F Bertram Diabetes . 2010,第10期

机译：Smad3抑制剂对内皮-间充质转化的阻断延迟了链脲佐菌素诱导的糖尿病性肾病的早期发展。
4. Study Mechanisms of Low Protein Diet Supplemented with Ketoanalogs on Reducing Proteinuria and Maintaining Nutritional Status in Type 2 Diabetic Nephropathy [C] . Ting Dong, Weijie Yuan, Jialin Wang, International Congress on Nutrition and Metabolism in Renal Disease . 2012

机译：酮甘露糖醛植物减少蛋白尿和维持2型糖尿病肾病营养状况的研究机制
5. Genetic and physiologic characterization of diabetic nephropathy in the type II diabetic nephropathy (T2DN) rat [D] . Schlick, Nancy Elizabeth 2006

机译：II型糖尿病肾病（T2DN）大鼠的糖尿病肾病的遗传和生理学表征
6. Endothelial–Mesenchymal Transition as a Novel Mechanism for Generating Myofibroblasts during Diabetic Nephropathy [O] . Akane Kizu, Damian Medici, Raghu Kalluri 2009

机译：内皮-间充质转化作为糖尿病肾病过程中生成成纤维细胞的新机制
7. Endothelial–Mesenchymal Transition as a Novel Mechanism for Generating Myofibroblasts during Diabetic Nephropathy [O] . Kizu, Akane, Medici, Damian, Kalluri, Raghu 2009

机译：内皮-间充质转化作为糖尿病肾病过程中生成成纤维细胞的新机制

Endothelial-Mesenchymal Transition as a Novel Mechanism for Generating Myofibroblasts during Diabetic Nephropathy

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