机译:特发性门静脉高压症中通过转化生长因子-{beta} 1 / Smad激活的内皮向间充质转变与门静脉狭窄相关。
From the Departments of Human Pathology,Kanazawa University Graduate School of Medicine, Kanazawa|Radiology,Kanazawa University Graduate School of Medicine, Kanazawa;
From the Departments of Human Pathology,Kanazawa University Graduate School of Medicine, Kanazawa;
From the Departments of Human Pathology,Kanazawa University Graduate School of Medicine, Kanazawa;
From the Departments of Human Pathology,Kanazawa University Graduate School of Medicine, Kanazawa;
From the Departments of Human Pathology,Kanazawa University Graduate School of Medicine, Kanazawa;
and the Departments of Hepatology,Osaka City University Graduate School of Medicine, Osaka, Japan;
and Nuclear Medicine,Osaka City University Graduate School of Medicine, Osaka, Japan;
and Gastroenterology,Kanazawa University Graduate School of Medicine, Kanazawa;
Radiology,Kanazawa University Graduate School of Medicine, Kanazawa;
From the Departments of Human Pathology,Kanazawa University Graduate School of Medicine, Kanazawa;
机译:特发性门静脉高压症中通过转化生长因子-beta1 / Smad激活的内皮向间充质转变与门静脉狭窄有关。
机译:转化生长因子-β1,转化生长因子-β2和转化生长因子-β3通过诱导Smad3依赖性上皮-间充质转化增强卵巢癌的转移潜能。
机译:转化生长因子-β2通过Smad依赖性和Smad依赖性信号传导的融合促进Snail介导的内皮-间充质转化
机译:转化生长因子-脽3的控制传递使间充质干细胞成软骨。
机译:在调节TGF-beta信号通路中转化生长因子-beta1激活和转化生长因子-beta受体2的表达水平。
机译:特发性门静脉高压症中通过转化生长因子-β1/ Smad激活的内皮向间充质转变与门静脉狭窄相关。
机译:特发性门静脉高压症中通过转化生长因子-β1/ Smad激活的内皮向间充质转变与门静脉狭窄相关。