Nickel-smelting fumes increased the expression of HIF-1α through PI3K/ERK pathway in NIH/3T3 cells

机译：镍熔炼烟通过NIH / 3T3细胞中的PI3K / ERK途径增加HIF-1α的表达

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摘要

Objective: The purpose of this study was to investigate the effects of Nickel (Ni) -smelting fumes on oncogenic proteins in vivo and in vitro.

机译：目的：本研究的目的是研究体内和体外镍熔炼烟气对致癌蛋白的影响。

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期刊名称 Journal of Occupational Health
作者
Dan Han; Yue Yang; Lin Zhang; Chao Wang; Yue Wang; Wen-Qiao Tan; Xue-Ying Hu; Yong-Hui Wu;
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作者单位

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年(卷),期 2016(58),5
年度 2016
页码 413–424
总页数 12
原文格式 PDF
正文语种
中图分类公共卫生工程;
关键词
Animal toxicity Cytotoxicity HIF-1α Kinase Nickel-smelting fumes;

机译：动物毒性;细胞毒性;HIF-1α;激酶;镍冶炼烟雾;

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专利

1. Effects of nickel-smelting fumes on the regulation of NIH/3T3 cell viability, necrosis, and expression of hMLH1 and RASSF1A. [J] . Jun Wang, Cui-Ping Yu, Xue-Ying Hu, Journal of environmental pathology, toxicology and oncology: official organ of the International Society for Environmental Toxicology and Cancer . 2014,第1期

机译：镍熔炼烟气对NIH / 3T3细胞活力，坏死以及hMLH1和RASSF1A表达的调节。
2. Nickel-smelting fumes induce mitochondrial damage and apoptosis, accompanied by decreases in viability, in NIH/3T3 cells [J] . Pan Yu-Lin, Xin Rui, Wang Sheng-Yuan, Archives of Biochemistry and Biophysics . 2018,第期

机译：镍冶炼烟雾诱导线粒体损伤和细胞凋亡，在NIH / 3T3细胞中伴随着活力的减少
3. Gadolinium-promoted cell cycle progression with enhanced S-phase entry via activation of both ERK and PI3K signaling pathways in NIH 3T3 cells [J] . Fu LJ, Li JX, Yang XG, Journal of biological inorganic chemistry: JBIC: a publication of the Society of Biological Inorganic Chemistry . 2009,第2期

机译：activation通过NIH 3T3细胞中ERK和PI3K信号通路的激活增强了S期进入，从而促进了细胞周期进程
4. Development of a Multidimensional ERLIC/IMAC/TiO2 Phosphoproteomic Method and Its Application to Kinase Pathway Analysis of PDGF-stimulated NIH 3T3 Cells [C] . Laura E. Herring, Kevin Blackburn, Kyle G. Grant, ASMS Conference on Mass Spectrometry and Allied Topics . 2014

机译：多维Erlic / IMAC / TiO2磷蛋白质方法的研制及其在PDGF刺激NIH 3T3细胞激酶途径分析中的应用
5. Vav3 and Rho GTPases, involved in the regulation of cell transformation, migration and invasion in NIH 3T3 fibroblasts and prostate cancer cells. [D] . Gong, Jianli. 2007

机译：Vav3和Rho GTPases参与NIH 3T3成纤维细胞和前列腺癌细胞的细胞转化，迁移和侵袭的调控。
6. Cytochalasin H Inhibits Angiogenesis via the Suppression of HIF-1α Protein Accumulation and VEGF Expression through PI3K/AKT/P70S6K and ERK1/2 Signaling Pathways in Non-Small Cell Lung Cancer Cells [O] . Yuefan Ma, Zihan Xiu, Zhiyuan Zhou, 2019

机译：细胞松弛素H通过非小细胞肺癌细胞中PI3K / AKT / P70S6K和ERK1 / 2信号通路抑制HIF-1α蛋白积累和VEGF表达抑制血管生成。
7. Cytochalasin H Inhibits Angiogenesis via the Suppression of HIF-1α Protein Accumulation and VEGF Expression through PI3K/AKT/P70S6K and ERK1/2 Signaling Pathways in Non-Small Cell Lung Cancer Cells [O] . Yuefan Ma, Zihan Xiu, Zhiyuan Zhou, 2019

机译：通过在非小细胞肺癌细胞中抑制HIF-1α蛋白质积累和VEGF表达，通过PI3K / AKT / P70S6K和ERK1 / 2信号传导途径抑制血管生成。

Nickel-smelting fumes increased the expression of HIF-1α through PI3K/ERK pathway in NIH/3T3 cells

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