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The Preliminary Study of Autophagy Induction of SA and MeSA by Confocal

机译:共聚焦诱导SA和MeSA自噬的初步研究

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Autophagy appears to be a highly conserved process from unicellular to multicellular eukaryotes which contributes to the equilibrium of intracelluar environment. While it would be harmful to the cells when it is excessive by inducing programmed cell death (PCD). It is a protein degradation process in which cells recycle cytoplasmic contents when subjected to environmental stress conditions or during certain stages of development. Previous studies have demonstrated autophagy can be induced during abiotic or biotic stresses, salicylic acid (SA) and methyl salicytic (MeSA) are endogenous signal molecules. We found SA and MeSA can induce autophagy in Arabidopsis thaliana respectively. While autophagy was not induced by SA or MeSA in tobacco suspension cells under the same concentration and period. The differences in stuctures or physiological states may contribute to the results.
机译:自噬似乎是从单细胞到多细胞真核生物的高度保守的过程,它有助于细胞内环境的平衡。通过过量导致程序性细胞死亡(PCD),过量时对细胞有害。这是一种蛋白质降解过程,其中细胞在受到环境压力条件或发育的某些阶段时会循环利用细胞质的内含物。先前的研究表明,在非生物或生物胁迫下可以诱导自噬,水杨酸(SA)和甲基水杨酸(MeSA)是内源性信号分子。我们发现SA和MeSA分别可以诱导拟南芥自噬。在相同浓度和时间下,SA或MeSA不会在烟草悬浮细胞中诱导自噬。结构或生理状态的差异可能有助于结果。

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